Matches in Nanopublications for { ?s ?p "[A number of rare mutations linked to familial AD (FAD) on the A? precursor protein (APP), Presenilin-1 (PS1), Presenilin- 2 (PS2), Adamalysin10, and other genetic risk factors for sporadic AD such as the ?4 allele of Apolipoprotein E (ApoE-?4) foster the accumulation of A? and also induce the entire spectrum of pathology associated with the disease.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- assertion description "[A number of rare mutations linked to familial AD (FAD) on the A? precursor protein (APP), Presenilin-1 (PS1), Presenilin- 2 (PS2), Adamalysin10, and other genetic risk factors for sporadic AD such as the ?4 allele of Apolipoprotein E (ApoE-?4) foster the accumulation of A? and also induce the entire spectrum of pathology associated with the disease.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[A number of rare mutations linked to familial AD (FAD) on the A? precursor protein (APP), Presenilin-1 (PS1), Presenilin- 2 (PS2), Adamalysin10, and other genetic risk factors for sporadic AD such as the ?4 allele of Apolipoprotein E (ApoE-?4) foster the accumulation of A? and also induce the entire spectrum of pathology associated with the disease.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[A number of rare mutations linked to familial AD (FAD) on the A? precursor protein (APP), Presenilin-1 (PS1), Presenilin- 2 (PS2), Adamalysin10, and other genetic risk factors for sporadic AD such as the ?4 allele of Apolipoprotein E (ApoE-?4) foster the accumulation of A? and also induce the entire spectrum of pathology associated with the disease.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP1249380.RAPE4dyY3FBeHWC4iIiWgjUTfdiAGaQZm9FIcDqHXBKjU130_assertion description "[A number of rare mutations linked to familial AD (FAD) on the A? precursor protein (APP), Presenilin-1 (PS1), Presenilin- 2 (PS2), Adamalysin10, and other genetic risk factors for sporadic AD such as the ?4 allele of Apolipoprotein E (ApoE-?4) foster the accumulation of A? and also induce the entire spectrum of pathology associated with the disease.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1249380.RAPE4dyY3FBeHWC4iIiWgjUTfdiAGaQZm9FIcDqHXBKjU130_provenance.
- NP1249377.RA-GS8JiqgU4EyZDykD2cCm6tlj9UrIdvnBmWj6_iqOgY130_assertion description "[A number of rare mutations linked to familial AD (FAD) on the A? precursor protein (APP), Presenilin-1 (PS1), Presenilin- 2 (PS2), Adamalysin10, and other genetic risk factors for sporadic AD such as the ?4 allele of Apolipoprotein E (ApoE-?4) foster the accumulation of A? and also induce the entire spectrum of pathology associated with the disease.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1249377.RA-GS8JiqgU4EyZDykD2cCm6tlj9UrIdvnBmWj6_iqOgY130_provenance.