Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP418281.RAOLGeayvgEuPKLirkZ7GERiqYCxrMD3-kRHz5DK7JVJo130_assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP418281.RAOLGeayvgEuPKLirkZ7GERiqYCxrMD3-kRHz5DK7JVJo130_provenance.
• assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP758517.RAcMkzm1Acpu18Pok20CPXt6A6-vSh5AHqEzD0JmdjaQw130_assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP758517.RAcMkzm1Acpu18Pok20CPXt6A6-vSh5AHqEzD0JmdjaQw130_provenance.
• assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP590218.RAcdhH7AjKG32-q_qCVCMbpAAMuYWSZ31LjugwnQDguD8130_assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP590218.RAcdhH7AjKG32-q_qCVCMbpAAMuYWSZ31LjugwnQDguD8130_provenance.
• NP917610.RAofiihY63RlOwb5cg2SY5ZsTYWrjbOFhTyR6gOka7tmU130_assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP917610.RAofiihY63RlOwb5cg2SY5ZsTYWrjbOFhTyR6gOka7tmU130_provenance.
• NP829354.RAOZ3RX6m4aq85gnXo15hqG5V9gYh5KiuPm198NZdU_k4130_assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP829354.RAOZ3RX6m4aq85gnXo15hqG5V9gYh5KiuPm198NZdU_k4130_provenance.
• NP590216.RAMKgzc69NNtn3jaT7kuLiss4NmgGZMgf-b_NVAP0rphA130_assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP590216.RAMKgzc69NNtn3jaT7kuLiss4NmgGZMgf-b_NVAP0rphA130_provenance.
• NP590215.RAxJYRqNIGfZbq6dtH5NjQJrwnsqrdzjkru8Zdq1MvYnM130_assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP590215.RAxJYRqNIGfZbq6dtH5NjQJrwnsqrdzjkru8Zdq1MvYnM130_provenance.
• NP590219.RA5IOSHNe8dU-vI2BuQLh_XC_7mkyM0zbladHkVcSQ0MQ130_assertion description "[AML1-TRPS1 stimulated proliferation of hematopoietic colony-forming cells and repressed the transcriptional activity of AML1 and GATA-1 by 2 different mechanisms: competition at their cognate DNA-binding sites and physical sequestrations of AML1 and GATA-1, suggesting that simultaneous deregulation of AML1 and GATA factors constitutes a basis for leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP590219.RA5IOSHNe8dU-vI2BuQLh_XC_7mkyM0zbladHkVcSQ0MQ130_provenance.