Matches in Nanopublications for { ?s ?p "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP782278.RAmrmp7VzGCVQIUp2YBFPN0ah5ZYUhA29e9YHCjbyiFww130_assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP782278.RAmrmp7VzGCVQIUp2YBFPN0ah5ZYUhA29e9YHCjbyiFww130_provenance.
- NP783926.RAVujdrlGHdo0hWmMq_tA_SDaS0GGC_DTuq1GLViUtSdE130_assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP783926.RAVujdrlGHdo0hWmMq_tA_SDaS0GGC_DTuq1GLViUtSdE130_provenance.
- NP783927.RAtfqgYN2J57m9Ky1GJQsPX64x0GInHoiwhT6TqFAcAok130_assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP783927.RAtfqgYN2J57m9Ky1GJQsPX64x0GInHoiwhT6TqFAcAok130_provenance.
- NP859898.RA4AbgQy5EUjIb3DorE4LNPSzzjAtTpv_oEBj7TuX5uUg130_assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP859898.RA4AbgQy5EUjIb3DorE4LNPSzzjAtTpv_oEBj7TuX5uUg130_provenance.
- NP877031.RA1g6pd0zQSt_TrzPm1IalVluaK0eBGVbt9xt2b7iPQwU130_assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP877031.RA1g6pd0zQSt_TrzPm1IalVluaK0eBGVbt9xt2b7iPQwU130_provenance.
- NP783925.RA2nnikMqbai_K3TCo0RKxO1SxPuxgeTJ5vJD9FD1nN7M130_assertion description "[Abrogation of the post-transcriptional regulation of c-Myc via methylation of a specific single CpG site in the TTP promoter presents a novel mechanism for the gain of selective resistance to the antiproliferative signaling of TGF-beta1 in HCC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP783925.RA2nnikMqbai_K3TCo0RKxO1SxPuxgeTJ5vJD9FD1nN7M130_provenance.