Matches in Nanopublications for { ?s ?p "[Because residual FAK protein in ISIS 15421-treated A549 cells was highly phosphorylated at the Tyr-397/Src homology (SH)2 binding site, expression of the FAK COOH-terminal domain (FRNK) was also used as an inhibitor of FAK function.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- assertion description "[Because residual FAK protein in ISIS 15421-treated A549 cells was highly phosphorylated at the Tyr-397/Src homology (SH)2 binding site, expression of the FAK COOH-terminal domain (FRNK) was also used as an inhibitor of FAK function.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP331890.RAVgGtfFrYXRfgfjA1Pu8KOR8EIOV2vXH_vE0ZCY7fAGM130_assertion description "[Because residual FAK protein in ISIS 15421-treated A549 cells was highly phosphorylated at the Tyr-397/Src homology (SH)2 binding site, expression of the FAK COOH-terminal domain (FRNK) was also used as an inhibitor of FAK function.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP331890.RAVgGtfFrYXRfgfjA1Pu8KOR8EIOV2vXH_vE0ZCY7fAGM130_provenance.
- NP659262.RABRKC-QdG28bJaCl5lXl_kcbvNIfyVu4xSZ0ZoM1GjMU130_assertion description "[Because residual FAK protein in ISIS 15421-treated A549 cells was highly phosphorylated at the Tyr-397/Src homology (SH)2 binding site, expression of the FAK COOH-terminal domain (FRNK) was also used as an inhibitor of FAK function.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP659262.RABRKC-QdG28bJaCl5lXl_kcbvNIfyVu4xSZ0ZoM1GjMU130_provenance.