Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP223722.RAl7HDbRsz2HjmNVH8MtZhrU2nl3WBrLwFIEEFd2NV4Us130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP223722.RAl7HDbRsz2HjmNVH8MtZhrU2nl3WBrLwFIEEFd2NV4Us130_provenance.
• NP691526.RAlNEeSakdiptaYtLWQcgvBVSAmAkHnPzujorl8kX_w8w130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP691526.RAlNEeSakdiptaYtLWQcgvBVSAmAkHnPzujorl8kX_w8w130_provenance.
• assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP477961.RAsBsiulO9LO33z_zc_WJCTPX2duNCiqVo8Q8f2aImy7I130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP477961.RAsBsiulO9LO33z_zc_WJCTPX2duNCiqVo8Q8f2aImy7I130_provenance.
• NP477963.RAgCLq8WwRMU9BcGLnDcriErnhZG2uqTTYh89Fx8Kvd5o130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP477963.RAgCLq8WwRMU9BcGLnDcriErnhZG2uqTTYh89Fx8Kvd5o130_provenance.
• NP608978.RAzlw6OBidvZOjScXol--k7EpwPzXf9dCCFyvytwj-tdQ130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP608978.RAzlw6OBidvZOjScXol--k7EpwPzXf9dCCFyvytwj-tdQ130_provenance.
• NP812816.RAzIm8VwlZxn-Dl3J4gw6Fm8MjJtWEWz-vQ2VovYtRluM130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP812816.RAzIm8VwlZxn-Dl3J4gw6Fm8MjJtWEWz-vQ2VovYtRluM130_provenance.
• NP986961.RA7AdqxCG7N1PjSyweNjZgnfvzYURxTGpXqae6DMN8dYk130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP986961.RA7AdqxCG7N1PjSyweNjZgnfvzYURxTGpXqae6DMN8dYk130_provenance.
• NP477959.RALgGlIQip-CiewyFy_6rVlBx0yLMCowSBViGkXrfDK14130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP477959.RALgGlIQip-CiewyFy_6rVlBx0yLMCowSBViGkXrfDK14130_provenance.
• NP477962.RALLhnajxTQtc5oXZN4GyXaDjOzMzupI48HIEnYR1nssQ130_assertion description "[Because the increased BACE1 activity observed in AD patients does not seem to arise from genetic mutations or polymorphisms in BACE1, the identification of PAR-4 as an endogenous regulator of BACE1 activity may have significant implications for developing novel therapeutic strategies for AD.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP477962.RALLhnajxTQtc5oXZN4GyXaDjOzMzupI48HIEnYR1nssQ130_provenance.