Matches in Nanopublications for { ?s ?p "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP240929.RAX17jYOUao8z0zx36-NIjvbTwraOIgdIlrdr8cpggBfg130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP240929.RAX17jYOUao8z0zx36-NIjvbTwraOIgdIlrdr8cpggBfg130_provenance.
- NP207306.RAqcueDhvFD7mUj3knm-lpEWP_9_-2ZNdycsMG-S7xeGA130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP207306.RAqcueDhvFD7mUj3knm-lpEWP_9_-2ZNdycsMG-S7xeGA130_provenance.
- NP620502.RAeQEHCVrR2vo8CAwaQ2obb0kEP9qKl2KBZfYCjzh5CEU130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP620502.RAeQEHCVrR2vo8CAwaQ2obb0kEP9qKl2KBZfYCjzh5CEU130_provenance.
- NP914004.RAnX-pG0FqpxPrrE-rag_L6a3LYWr-os0Bj3KckFuB2Ug130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP914004.RAnX-pG0FqpxPrrE-rag_L6a3LYWr-os0Bj3KckFuB2Ug130_provenance.
- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP222461.RAYG_joV4Ef9CXEnTVj5SWs_cQuAA47Wn05s4uXNohFR4130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP222461.RAYG_joV4Ef9CXEnTVj5SWs_cQuAA47Wn05s4uXNohFR4130_provenance.
- NP374933.RAbn0b-Le2ZCbecl0oPFT4I4tXZ9sG6ZWTaj8ohkw1V8M130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP374933.RAbn0b-Le2ZCbecl0oPFT4I4tXZ9sG6ZWTaj8ohkw1V8M130_provenance.
- NP702474.RAfVFUl-7DV4dECkufb5YADsxtSn8tSejTBVDNru52Ayc130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP702474.RAfVFUl-7DV4dECkufb5YADsxtSn8tSejTBVDNru52Ayc130_provenance.
- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP548296.RAV-n8J1CJGtaAjfC6NlEfbcK7P322oKAd86nCphantdg130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548296.RAV-n8J1CJGtaAjfC6NlEfbcK7P322oKAd86nCphantdg130_provenance.
- NP374783.RAtHP6NyWL2D2SWdlgG4mjopMM57DNRXJDNpHD1Fygv2U130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP374783.RAtHP6NyWL2D2SWdlgG4mjopMM57DNRXJDNpHD1Fygv2U130_provenance.
- NP548295.RAkRzXWcmRede6TeGETCiTYQ1MHLMjLJGTVBTIpzHM1-Q130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548295.RAkRzXWcmRede6TeGETCiTYQ1MHLMjLJGTVBTIpzHM1-Q130_provenance.
- NP548297.RAjKTQbOoOTLN40AZ1OTtWb7wbzvoyD0_BE522Ivdm4Ps130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548297.RAjKTQbOoOTLN40AZ1OTtWb7wbzvoyD0_BE522Ivdm4Ps130_provenance.
- NP702353.RANychMTf-uOCQpQ0YUACanL7AFgiZdhyTTEVsmJZ13BQ130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP702353.RANychMTf-uOCQpQ0YUACanL7AFgiZdhyTTEVsmJZ13BQ130_provenance.
- NP548289.RA9s6gWabzKOl3TDg9SAWqaZx9NFhkxQxkfF4BqDcZ5UQ130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548289.RA9s6gWabzKOl3TDg9SAWqaZx9NFhkxQxkfF4BqDcZ5UQ130_provenance.
- NP548290.RA-imBMORCJlpsATJ_hWCdjgq2qQLD7Riywl32ncRRX5o130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548290.RA-imBMORCJlpsATJ_hWCdjgq2qQLD7Riywl32ncRRX5o130_provenance.
- NP548291.RA4x3chV27iPgCh0V4uUhLW3_hz0pTBpM6A69i8hqObcg130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548291.RA4x3chV27iPgCh0V4uUhLW3_hz0pTBpM6A69i8hqObcg130_provenance.
- NP548292.RA167UGIFLroLu3Id5IrdiH4GySpwt7JYGnUd_Z2EAnXU130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548292.RA167UGIFLroLu3Id5IrdiH4GySpwt7JYGnUd_Z2EAnXU130_provenance.
- NP548293.RA1Qrka0r0hzg8UNII2NiPO39HzPx-uHCd6Y8v1l3mdLU130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548293.RA1Qrka0r0hzg8UNII2NiPO39HzPx-uHCd6Y8v1l3mdLU130_provenance.
- NP548294.RA2iOFvjkWF4rld8h4cTRv5HNKAO4rJFBWvWWdmwbvOn4130_assertion description "[Defective TGF-beta signaling in endothelial cells attributable to mutations in endoglin or the type I receptor ALK-1 leads to hereditary hemorrhagic telangiectasia, whereas defective BMP signaling attributable to mutations in the BMP receptor II has been associated with development of primary pulmonary hypertension.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP548294.RA2iOFvjkWF4rld8h4cTRv5HNKAO4rJFBWvWWdmwbvOn4130_provenance.