Matches in Nanopublications for { ?s ?p "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP617281.RATJd5tuZmbh82ruy8sjnjGq1IrWvNF8mT31bRFWfPdlQ130_assertion description "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP617281.RATJd5tuZmbh82ruy8sjnjGq1IrWvNF8mT31bRFWfPdlQ130_provenance.
- NP829771.RAaNGUVkOvPXV0dxpauObX0kZGVSOxyBVdotbvTCRBLAI130_assertion description "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP829771.RAaNGUVkOvPXV0dxpauObX0kZGVSOxyBVdotbvTCRBLAI130_provenance.
- NP654954.RAsSpywTR0NtToymc8WhFQS0VIt3r84zsR1FnzRzp4CK4130_assertion description "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP654954.RAsSpywTR0NtToymc8WhFQS0VIt3r84zsR1FnzRzp4CK4130_provenance.
- NP357242.RANsY9by6zDgZVmXSYsYP9KWTGU7_2GFl5-qeaqDeoA-0130_assertion description "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP357242.RANsY9by6zDgZVmXSYsYP9KWTGU7_2GFl5-qeaqDeoA-0130_provenance.
- NP357582.RAPIoTqN5G538abj9cQnd7-VuYq9vXXrQd3PoX77MkrF4130_assertion description "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP357582.RAPIoTqN5G538abj9cQnd7-VuYq9vXXrQd3PoX77MkrF4130_provenance.
- NP617630.RAPjPibWdAv9JfeP5ZFACvaivg0z8f3SG3bEFfOrxD_dk130_assertion description "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP617630.RAPjPibWdAv9JfeP5ZFACvaivg0z8f3SG3bEFfOrxD_dk130_provenance.
- NP829504.RAJG8W08WWB1lWMfq5Kz-7_Z5tx38zqLb15NR-XWCMyKA130_assertion description "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP829504.RAJG8W08WWB1lWMfq5Kz-7_Z5tx38zqLb15NR-XWCMyKA130_provenance.
- NP653715.RA174H8JXDTJ_nlb6_ZhUB1fzjQo8LgMO8eaHhdCXh1KA130_assertion description "[EC is known for extensive epigenetic alterations, including overexpression of HDAC and DNMT enzymes, and the frequent epigenetic silencing of DNA repair genes such as MLH1, tumor suppressor genes PTEN, and progesterone receptor, which suggests a potentially high sensitivity of this type of cancer to HDAC inhibitors.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP653715.RA174H8JXDTJ_nlb6_ZhUB1fzjQo8LgMO8eaHhdCXh1KA130_provenance.