Matches in Nanopublications for { ?s ?p "[Even though the PAD2 knockout mice displayed a dramatic reduction in the amount of citrullination present in the CNS, indicating that PAD2 is indeed responsible for the majority of detectable citrullination observed in EAE, the development of EAE was not impaired by genetic deletion of PAD2, suggesting that PAD2 catalyzed citrullination is not essential to the development of EAE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP161739.RA93bXYL-wXcOaKMV-aWIMQe09gY0kNlkGkPMXCXm8VOA130_assertion description "[Even though the PAD2 knockout mice displayed a dramatic reduction in the amount of citrullination present in the CNS, indicating that PAD2 is indeed responsible for the majority of detectable citrullination observed in EAE, the development of EAE was not impaired by genetic deletion of PAD2, suggesting that PAD2 catalyzed citrullination is not essential to the development of EAE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP161739.RA93bXYL-wXcOaKMV-aWIMQe09gY0kNlkGkPMXCXm8VOA130_provenance.
- assertion description "[Even though the PAD2 knockout mice displayed a dramatic reduction in the amount of citrullination present in the CNS, indicating that PAD2 is indeed responsible for the majority of detectable citrullination observed in EAE, the development of EAE was not impaired by genetic deletion of PAD2, suggesting that PAD2 catalyzed citrullination is not essential to the development of EAE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP560612.RAgERRDAoKGtrK8KlJyPL-ykZaT68_FnE-l89Y7X9lWJc130_assertion description "[Even though the PAD2 knockout mice displayed a dramatic reduction in the amount of citrullination present in the CNS, indicating that PAD2 is indeed responsible for the majority of detectable citrullination observed in EAE, the development of EAE was not impaired by genetic deletion of PAD2, suggesting that PAD2 catalyzed citrullination is not essential to the development of EAE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP560612.RAgERRDAoKGtrK8KlJyPL-ykZaT68_FnE-l89Y7X9lWJc130_provenance.
- NP863439.RAFv02l-v8CvxGg0EdngBXrfnsUdtkEYBzWzGabUmIEZc130_assertion description "[Even though the PAD2 knockout mice displayed a dramatic reduction in the amount of citrullination present in the CNS, indicating that PAD2 is indeed responsible for the majority of detectable citrullination observed in EAE, the development of EAE was not impaired by genetic deletion of PAD2, suggesting that PAD2 catalyzed citrullination is not essential to the development of EAE.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP863439.RAFv02l-v8CvxGg0EdngBXrfnsUdtkEYBzWzGabUmIEZc130_provenance.