Matches in Nanopublications for { ?s ?p "[Furthermore, Fhit-deficient mice demonstrated a decrease in the number of pChk2-positive cells not only in dysplastic lesions but also in N-nitrosobenzylamine-induced papilloma of the forestomach, suggesting that lack of Fhit expression and the resultant defects of the ataxia telangiectasia mutated-Chk2 pathway can cause a difference in the incidence of N-nitrosobenzylamine-induced forestomach lesions.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP299221.RAZBXF0yLRvVchmj95pWfzjp95S1qKpAoVEkPqlypTV44130_assertion description "[Furthermore, Fhit-deficient mice demonstrated a decrease in the number of pChk2-positive cells not only in dysplastic lesions but also in N-nitrosobenzylamine-induced papilloma of the forestomach, suggesting that lack of Fhit expression and the resultant defects of the ataxia telangiectasia mutated-Chk2 pathway can cause a difference in the incidence of N-nitrosobenzylamine-induced forestomach lesions.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP299221.RAZBXF0yLRvVchmj95pWfzjp95S1qKpAoVEkPqlypTV44130_provenance.
- assertion description "[Furthermore, Fhit-deficient mice demonstrated a decrease in the number of pChk2-positive cells not only in dysplastic lesions but also in N-nitrosobenzylamine-induced papilloma of the forestomach, suggesting that lack of Fhit expression and the resultant defects of the ataxia telangiectasia mutated-Chk2 pathway can cause a difference in the incidence of N-nitrosobenzylamine-induced forestomach lesions.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP647804.RAv4A-LUkN9zK7v8YeKDGCcH_0uIl1bEwkVKvlgYBeV_Q130_assertion description "[Furthermore, Fhit-deficient mice demonstrated a decrease in the number of pChk2-positive cells not only in dysplastic lesions but also in N-nitrosobenzylamine-induced papilloma of the forestomach, suggesting that lack of Fhit expression and the resultant defects of the ataxia telangiectasia mutated-Chk2 pathway can cause a difference in the incidence of N-nitrosobenzylamine-induced forestomach lesions.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP647804.RAv4A-LUkN9zK7v8YeKDGCcH_0uIl1bEwkVKvlgYBeV_Q130_provenance.
- NP402964.RA1iJL6IqXc58_7-0dVltEeMQ4hPXMr_go6xtrMP3i-FA130_assertion description "[Furthermore, Fhit-deficient mice demonstrated a decrease in the number of pChk2-positive cells not only in dysplastic lesions but also in N-nitrosobenzylamine-induced papilloma of the forestomach, suggesting that lack of Fhit expression and the resultant defects of the ataxia telangiectasia mutated-Chk2 pathway can cause a difference in the incidence of N-nitrosobenzylamine-induced forestomach lesions.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP402964.RA1iJL6IqXc58_7-0dVltEeMQ4hPXMr_go6xtrMP3i-FA130_provenance.