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Matches in Nanopublications for { ?s ?p "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP226040.RAsOJx9x3EgcVXRFoTNrxkKU9fANNH3wd0djgycQzOo0M130_assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP226040.RAsOJx9x3EgcVXRFoTNrxkKU9fANNH3wd0djgycQzOo0M130_provenance.
• NP172923.RA0aAgXM8G9zmYjF0VR0fvD4_yO1KEqMSI3M5A6WX63Bs130_assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP172923.RA0aAgXM8G9zmYjF0VR0fvD4_yO1KEqMSI3M5A6WX63Bs130_provenance.
• assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP950650.RAQclWfjn4Qbhta0sP71f2SSLxDYaSFfxCRikfOpK1_p4130_assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP950650.RAQclWfjn4Qbhta0sP71f2SSLxDYaSFfxCRikfOpK1_p4130_provenance.
• assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP607619.RAg7NjJQz8Ltk6h5K3dGe3yqEK6lnl0SpG2ZoXwOnFtyY130_assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP607619.RAg7NjJQz8Ltk6h5K3dGe3yqEK6lnl0SpG2ZoXwOnFtyY130_provenance.
• NP997894.RAJsaY17mzrC5Rxoh3YU42hxNaEahw1OdE1kN0SEVRcX0130_assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP997894.RAJsaY17mzrC5Rxoh3YU42hxNaEahw1OdE1kN0SEVRcX0130_provenance.
• NP950648.RANHsXsIc0M8JcEk-o55zX8cU3WobCVfI3wVSqkY4JXOc130_assertion description "[Genetic loss of PAI-1 in mdx dystrophic mice anticipated muscle fibrosis through these sequential mechanisms: the alteration of collagen metabolism by uPA-mediated proteolytic processing of transforming growth factor (TGF)-? in muscle fibroblasts and the activation of miR-21 expression, which inhibited phosphatase and tensin homologue and enhanced AKT signaling, thus endowing TGF-? with a remarkable cell proliferation-promoting potential.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP950648.RANHsXsIc0M8JcEk-o55zX8cU3WobCVfI3wVSqkY4JXOc130_provenance.