Matches in Nanopublications for { ?s ?p "[Genetic studies in humans have shown that large intronic deletions in WNK1 lead to its overexpression and are responsible for pseudohypoaldosteronism type II, an autosomal dominant disorder characterized by hypertension, increased renal salt reabsorption, and impaired K+ and H+ excretion.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP368338.RATYd4xTfLqRlays3qsty6SaTSSjead4ebrXI4mZoTvmY130_assertion description "[Genetic studies in humans have shown that large intronic deletions in WNK1 lead to its overexpression and are responsible for pseudohypoaldosteronism type II, an autosomal dominant disorder characterized by hypertension, increased renal salt reabsorption, and impaired K+ and H+ excretion.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP368338.RATYd4xTfLqRlays3qsty6SaTSSjead4ebrXI4mZoTvmY130_provenance.
- assertion description "[Genetic studies in humans have shown that large intronic deletions in WNK1 lead to its overexpression and are responsible for pseudohypoaldosteronism type II, an autosomal dominant disorder characterized by hypertension, increased renal salt reabsorption, and impaired K+ and H+ excretion.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Genetic studies in humans have shown that large intronic deletions in WNK1 lead to its overexpression and are responsible for pseudohypoaldosteronism type II, an autosomal dominant disorder characterized by hypertension, increased renal salt reabsorption, and impaired K+ and H+ excretion.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Genetic studies in humans have shown that large intronic deletions in WNK1 lead to its overexpression and are responsible for pseudohypoaldosteronism type II, an autosomal dominant disorder characterized by hypertension, increased renal salt reabsorption, and impaired K+ and H+ excretion.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP420078.RAUn5kriG38WzlFhQbw0tX7maUOU93UiCYDFXIxrPeXR8130_assertion description "[Genetic studies in humans have shown that large intronic deletions in WNK1 lead to its overexpression and are responsible for pseudohypoaldosteronism type II, an autosomal dominant disorder characterized by hypertension, increased renal salt reabsorption, and impaired K+ and H+ excretion.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP420078.RAUn5kriG38WzlFhQbw0tX7maUOU93UiCYDFXIxrPeXR8130_provenance.
- NP420079.RAGD5ku3hBWQPl8-M36FeDPeU37JoXj_FjoYLSk4GqImY130_assertion description "[Genetic studies in humans have shown that large intronic deletions in WNK1 lead to its overexpression and are responsible for pseudohypoaldosteronism type II, an autosomal dominant disorder characterized by hypertension, increased renal salt reabsorption, and impaired K+ and H+ excretion.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP420079.RAGD5ku3hBWQPl8-M36FeDPeU37JoXj_FjoYLSk4GqImY130_provenance.