Matches in Nanopublications for { ?s ?p "[HER2 overexpression is a hallmark of a particularly aggressive subset of breast tumors, and its activation is strictly dependent on the trans-interaction with other members of HER family; in particular, the activation of the PI3K/Akt survival pathway, so critically important in tumorigenesis, is predominantly driven through phosphorylation of the kinase-inactive member HER3.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- assertion description "[HER2 overexpression is a hallmark of a particularly aggressive subset of breast tumors, and its activation is strictly dependent on the trans-interaction with other members of HER family; in particular, the activation of the PI3K/Akt survival pathway, so critically important in tumorigenesis, is predominantly driven through phosphorylation of the kinase-inactive member HER3.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[HER2 overexpression is a hallmark of a particularly aggressive subset of breast tumors, and its activation is strictly dependent on the trans-interaction with other members of HER family; in particular, the activation of the PI3K/Akt survival pathway, so critically important in tumorigenesis, is predominantly driven through phosphorylation of the kinase-inactive member HER3.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP865447.RA2MMkBbbvBAdNaoVN-U8_DIxBHkeSWvtoWv-irfNnr8I130_assertion description "[HER2 overexpression is a hallmark of a particularly aggressive subset of breast tumors, and its activation is strictly dependent on the trans-interaction with other members of HER family; in particular, the activation of the PI3K/Akt survival pathway, so critically important in tumorigenesis, is predominantly driven through phosphorylation of the kinase-inactive member HER3.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP865447.RA2MMkBbbvBAdNaoVN-U8_DIxBHkeSWvtoWv-irfNnr8I130_provenance.
- NP725167.RAJeFs8KMa3HsLgiugfCjVLWE6cg-Yi5NPFLkDVZfbOiU130_assertion description "[HER2 overexpression is a hallmark of a particularly aggressive subset of breast tumors, and its activation is strictly dependent on the trans-interaction with other members of HER family; in particular, the activation of the PI3K/Akt survival pathway, so critically important in tumorigenesis, is predominantly driven through phosphorylation of the kinase-inactive member HER3.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP725167.RAJeFs8KMa3HsLgiugfCjVLWE6cg-Yi5NPFLkDVZfbOiU130_provenance.
- NP725171.RAJQrsKlq_SkdXqvvz1kM3RgyxR_bMsQTOj0D8ReFsIx0130_assertion description "[HER2 overexpression is a hallmark of a particularly aggressive subset of breast tumors, and its activation is strictly dependent on the trans-interaction with other members of HER family; in particular, the activation of the PI3K/Akt survival pathway, so critically important in tumorigenesis, is predominantly driven through phosphorylation of the kinase-inactive member HER3.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP725171.RAJQrsKlq_SkdXqvvz1kM3RgyxR_bMsQTOj0D8ReFsIx0130_provenance.