Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP826670.RABEYxhhLFrKVTP5mppLNDaw9EZPsSfzY_PUwMyAATM7w130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP826670.RABEYxhhLFrKVTP5mppLNDaw9EZPsSfzY_PUwMyAATM7w130_provenance.
• assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP451759.RAXkJhEOUvwDuUKAWe5vONAyZgxLcRzBmxLmrM39D46KU130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP451759.RAXkJhEOUvwDuUKAWe5vONAyZgxLcRzBmxLmrM39D46KU130_provenance.
• NP451763.RAbCi9SYu-rrEFVCFxIp9nDebBD-xBz1JeTAgGD9trZHk130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP451763.RAbCi9SYu-rrEFVCFxIp9nDebBD-xBz1JeTAgGD9trZHk130_provenance.
• NP451761.RAmHw-S0XU7ZvVE1p5_bOeoJULcwqVhpJcasztkOOgWug130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP451761.RAmHw-S0XU7ZvVE1p5_bOeoJULcwqVhpJcasztkOOgWug130_provenance.
• NP582831.RAH5psmsg4eqTcC1Lz5F5zpBUaJcb4HtnNAVyA9AHgB1s130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP582831.RAH5psmsg4eqTcC1Lz5F5zpBUaJcb4HtnNAVyA9AHgB1s130_provenance.
• NP582826.RA58YJS52cBCdCJ9J4vxauJP0KMuLOGZusvzjcA-wkBvc130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP582826.RA58YJS52cBCdCJ9J4vxauJP0KMuLOGZusvzjcA-wkBvc130_provenance.
• NP582894.RA-9DFyDzi0yF1lLt3JzGnEyG0uqmap7gZK8-OtMIQhkc130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP582894.RA-9DFyDzi0yF1lLt3JzGnEyG0uqmap7gZK8-OtMIQhkc130_provenance.
• NP741425.RAJvDBTtbHRv05JkL0gRLOR4RmMRBrqehEIWFVCaapHlI130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP741425.RAJvDBTtbHRv05JkL0gRLOR4RmMRBrqehEIWFVCaapHlI130_provenance.
• NP451762.RAwV3MuS0gXl4gt7aWPvkpucQEqY5GYRCP348-vPzqVkM130_assertion description "[Here, we demonstrate that genetic deficiency of TLR4 or MyD88 is associated with a significant reduction of aortic plaque areas in atherosclerosis-prone apolipoprotein E-deficient mice, despite persistent hypercholesterolemia, implying an important role for the innate immune system in atherogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP451762.RAwV3MuS0gXl4gt7aWPvkpucQEqY5GYRCP348-vPzqVkM130_provenance.