Matches in Nanopublications for { ?s ?p "[IL28B polymorphism seems to be involved in the development of HCV-induced HCC and in the course of HCV recurrence after LT. T allele may be regarded as a genetic risk factor for HCV-related carcinogenesis, posttransplant fibrosis progression, and antiviral therapy failure.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP660582.RAkOEt5fDkvYyWzDrftKHZReThuNvtkZq7ZNDrXlAyqn4130_assertion description "[IL28B polymorphism seems to be involved in the development of HCV-induced HCC and in the course of HCV recurrence after LT. T allele may be regarded as a genetic risk factor for HCV-related carcinogenesis, posttransplant fibrosis progression, and antiviral therapy failure.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP660582.RAkOEt5fDkvYyWzDrftKHZReThuNvtkZq7ZNDrXlAyqn4130_provenance.
- NP432153.RAQ6YubIFd7nBIAupiPV0U1V4JPBRGPnnvVFeKQsJ-2a8130_assertion description "[IL28B polymorphism seems to be involved in the development of HCV-induced HCC and in the course of HCV recurrence after LT. T allele may be regarded as a genetic risk factor for HCV-related carcinogenesis, posttransplant fibrosis progression, and antiviral therapy failure.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP432153.RAQ6YubIFd7nBIAupiPV0U1V4JPBRGPnnvVFeKQsJ-2a8130_provenance.
- NP983258.RAQA8gjnbLi6zPWN7h8YFmnlDKnzorUSoQLAEv9AL-Acc130_assertion description "[IL28B polymorphism seems to be involved in the development of HCV-induced HCC and in the course of HCV recurrence after LT. T allele may be regarded as a genetic risk factor for HCV-related carcinogenesis, posttransplant fibrosis progression, and antiviral therapy failure.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP983258.RAQA8gjnbLi6zPWN7h8YFmnlDKnzorUSoQLAEv9AL-Acc130_provenance.
- assertion description "[IL28B polymorphism seems to be involved in the development of HCV-induced HCC and in the course of HCV recurrence after LT. T allele may be regarded as a genetic risk factor for HCV-related carcinogenesis, posttransplant fibrosis progression, and antiviral therapy failure.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP967152.RAsTAvQ3CJx1q6BOdSes4SLx6DQ5lPp7rfn1-KQx0UOyc130_assertion description "[IL28B polymorphism seems to be involved in the development of HCV-induced HCC and in the course of HCV recurrence after LT. T allele may be regarded as a genetic risk factor for HCV-related carcinogenesis, posttransplant fibrosis progression, and antiviral therapy failure.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP967152.RAsTAvQ3CJx1q6BOdSes4SLx6DQ5lPp7rfn1-KQx0UOyc130_provenance.
- NP983265.RAGkBxAUMBWFGkOVr2Ey5FN7uBSZq7c_g-mqxdSPOwGTw130_assertion description "[IL28B polymorphism seems to be involved in the development of HCV-induced HCC and in the course of HCV recurrence after LT. T allele may be regarded as a genetic risk factor for HCV-related carcinogenesis, posttransplant fibrosis progression, and antiviral therapy failure.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP983265.RAGkBxAUMBWFGkOVr2Ey5FN7uBSZq7c_g-mqxdSPOwGTw130_provenance.