Matches in Nanopublications for { ?s ?p "[In addition to its reported role in promoting HBV replication, the binding of HBx to DDB1 may induce genetic instability in regenerating hepatocytes and thereby contribute to HCC development, thus making this HBV-host protein interaction an attractive target for new therapeutic intervention.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP856090.RAo_2sHE0tBsc122yQjqqbQspR0QzP9ZzsAEJVeTd3s_M130_assertion description "[In addition to its reported role in promoting HBV replication, the binding of HBx to DDB1 may induce genetic instability in regenerating hepatocytes and thereby contribute to HCC development, thus making this HBV-host protein interaction an attractive target for new therapeutic intervention.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP856090.RAo_2sHE0tBsc122yQjqqbQspR0QzP9ZzsAEJVeTd3s_M130_provenance.
- NP398627.RA12i8p5CDhklTdUQUv90v_h7jSzATOCgEW_6nSzDAshs130_assertion description "[In addition to its reported role in promoting HBV replication, the binding of HBx to DDB1 may induce genetic instability in regenerating hepatocytes and thereby contribute to HCC development, thus making this HBV-host protein interaction an attractive target for new therapeutic intervention.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP398627.RA12i8p5CDhklTdUQUv90v_h7jSzATOCgEW_6nSzDAshs130_provenance.
- assertion description "[In addition to its reported role in promoting HBV replication, the binding of HBx to DDB1 may induce genetic instability in regenerating hepatocytes and thereby contribute to HCC development, thus making this HBV-host protein interaction an attractive target for new therapeutic intervention.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP350786.RAf2NTgoi9kwr6byQL8wIY9j1a8uQvn-eLU-gdM45ltgM130_assertion description "[In addition to its reported role in promoting HBV replication, the binding of HBx to DDB1 may induce genetic instability in regenerating hepatocytes and thereby contribute to HCC development, thus making this HBV-host protein interaction an attractive target for new therapeutic intervention.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP350786.RAf2NTgoi9kwr6byQL8wIY9j1a8uQvn-eLU-gdM45ltgM130_provenance.
- NP692251.RAsIVJi3lKrZ63x_bGDOwy5h2d7CelAg6XDVWqbBDXhkI130_assertion description "[In addition to its reported role in promoting HBV replication, the binding of HBx to DDB1 may induce genetic instability in regenerating hepatocytes and thereby contribute to HCC development, thus making this HBV-host protein interaction an attractive target for new therapeutic intervention.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP692251.RAsIVJi3lKrZ63x_bGDOwy5h2d7CelAg6XDVWqbBDXhkI130_provenance.