Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP785298.RAd6BrYHU1langS7aPNBsbXcVkrIQLJrHcaq3WTBMJIvA130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP785298.RAd6BrYHU1langS7aPNBsbXcVkrIQLJrHcaq3WTBMJIvA130_provenance.
• assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP231952.RACuHBgkIFl30-tZfTA_nN2G9Tif_ok-NmncDjXLUA8RY130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP231952.RACuHBgkIFl30-tZfTA_nN2G9Tif_ok-NmncDjXLUA8RY130_provenance.
• NP1124895.RAab_XiAlEcoGQ8V2DHw1SYNip5oezjM0X-0RXBSR3v_M130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1124895.RAab_XiAlEcoGQ8V2DHw1SYNip5oezjM0X-0RXBSR3v_M130_provenance.
• NP1124897.RAUpE9OdkPmKe41MuNE7fGPTWemJKx3kn0e3I5-qMutNI130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1124897.RAUpE9OdkPmKe41MuNE7fGPTWemJKx3kn0e3I5-qMutNI130_provenance.
• NP1124901.RAYis_TEBmPujLrk2nnjdMTa99zh9-Vx5YWaW0Fp59BQs130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1124901.RAYis_TEBmPujLrk2nnjdMTa99zh9-Vx5YWaW0Fp59BQs130_provenance.
• NP645085.RAiHpuQ4wZOPrssMr7jWYID25wLsCfvlU3pM1wdztlvHg130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP645085.RAiHpuQ4wZOPrssMr7jWYID25wLsCfvlU3pM1wdztlvHg130_provenance.
• NP232802.RA2rk5a8zp5W27UDMhbMQxTEyw9sTwGJzSTKAph1zAbpM130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP232802.RA2rk5a8zp5W27UDMhbMQxTEyw9sTwGJzSTKAph1zAbpM130_provenance.
• NP644812.RAGqXcfGXsNUgbrmtuPxyNzE0IjZz_6xDWxhG2suMm0JA130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP644812.RAGqXcfGXsNUgbrmtuPxyNzE0IjZz_6xDWxhG2suMm0JA130_provenance.
• NP784529.RA1K559dKa9ee0xyC8siJNy0N76jGnecHL-mKRMnTeTnE130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP784529.RA1K559dKa9ee0xyC8siJNy0N76jGnecHL-mKRMnTeTnE130_provenance.
• NP1124896.RA2uF1-8Fah6NrcV7uvG7d3oga88yQiOds-xR03MuMcMY130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1124896.RA2uF1-8Fah6NrcV7uvG7d3oga88yQiOds-xR03MuMcMY130_provenance.
• NP1124899.RA_uC8E-fwh0L52IWcsEwoyKux_tPmbJD78Cgm7hfCqyI130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1124899.RA_uC8E-fwh0L52IWcsEwoyKux_tPmbJD78Cgm7hfCqyI130_provenance.
• NP1124900.RA4LJkR4yGVrEF7a2zccgsG86WMZAE_29FcWCyEu3UBZI130_assertion description "[In conclusion, our data revealed that miR-126 functions as a tumor suppressor in CRC cells by regulating CXCR4 expression via the AKT and ERK1/2 signaling pathways and might be a novel target for therapeutic strategies in CRC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1124900.RA4LJkR4yGVrEF7a2zccgsG86WMZAE_29FcWCyEu3UBZI130_provenance.