Matches in Nanopublications for { ?s ?p "[In conclusion, our results indicated for the first time a regulatory role of ER-?36 in up-regulation of EGFR expression and down-regulation of ER-?66 expression, which could be an underlying mechanism for the growth status switch in breast tumors that contribute to the generation of acquired TAM resistance.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP718955.RAiJkhIMr-uRN3jCLq3cMDck6gPoqRe5YQIYrDLHro-2E130_assertion description "[In conclusion, our results indicated for the first time a regulatory role of ER-?36 in up-regulation of EGFR expression and down-regulation of ER-?66 expression, which could be an underlying mechanism for the growth status switch in breast tumors that contribute to the generation of acquired TAM resistance.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP718955.RAiJkhIMr-uRN3jCLq3cMDck6gPoqRe5YQIYrDLHro-2E130_provenance.
- assertion description "[In conclusion, our results indicated for the first time a regulatory role of ER-?36 in up-regulation of EGFR expression and down-regulation of ER-?66 expression, which could be an underlying mechanism for the growth status switch in breast tumors that contribute to the generation of acquired TAM resistance.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[In conclusion, our results indicated for the first time a regulatory role of ER-?36 in up-regulation of EGFR expression and down-regulation of ER-?66 expression, which could be an underlying mechanism for the growth status switch in breast tumors that contribute to the generation of acquired TAM resistance.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP1060883.RAeONQcB49Vt2ow3a9qscQIx7z3Y3vOmM4sxvOTeBAt3Q130_assertion description "[In conclusion, our results indicated for the first time a regulatory role of ER-?36 in up-regulation of EGFR expression and down-regulation of ER-?66 expression, which could be an underlying mechanism for the growth status switch in breast tumors that contribute to the generation of acquired TAM resistance.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1060883.RAeONQcB49Vt2ow3a9qscQIx7z3Y3vOmM4sxvOTeBAt3Q130_provenance.
- NP370453.RA9Xe3bGD2EKwnZn52_Cm5BL33zcr6shUG3puKMBa3ZwI130_assertion description "[In conclusion, our results indicated for the first time a regulatory role of ER-?36 in up-regulation of EGFR expression and down-regulation of ER-?66 expression, which could be an underlying mechanism for the growth status switch in breast tumors that contribute to the generation of acquired TAM resistance.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP370453.RA9Xe3bGD2EKwnZn52_Cm5BL33zcr6shUG3puKMBa3ZwI130_provenance.
- NP1060882.RA270w0lM1xTVEJQ4AX9NTNdDM3Qi-kow6afHwbSsYphQ130_assertion description "[In conclusion, our results indicated for the first time a regulatory role of ER-?36 in up-regulation of EGFR expression and down-regulation of ER-?66 expression, which could be an underlying mechanism for the growth status switch in breast tumors that contribute to the generation of acquired TAM resistance.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1060882.RA270w0lM1xTVEJQ4AX9NTNdDM3Qi-kow6afHwbSsYphQ130_provenance.