Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP781557.RAnjkoo75oeN073SD4Wfr9yt-UkktWTa9IfxIkvL1ez1k130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP781557.RAnjkoo75oeN073SD4Wfr9yt-UkktWTa9IfxIkvL1ez1k130_provenance.
• NP772491.RAWqXRjM9s-nb1rxhUmRmlyFWuLQveEJGxl2ecptqJgOE130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772491.RAWqXRjM9s-nb1rxhUmRmlyFWuLQveEJGxl2ecptqJgOE130_provenance.
• NP772492.RAe8r4tB3viDsSmtEGp743ZvLGeiSXSo4h4B1LiVvmQjM130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772492.RAe8r4tB3viDsSmtEGp743ZvLGeiSXSo4h4B1LiVvmQjM130_provenance.
• NP772500.RAf75qCMVNoC2qJ3dZKhOe59k4EIVdkZffpwqoRV_UP_Q130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772500.RAf75qCMVNoC2qJ3dZKhOe59k4EIVdkZffpwqoRV_UP_Q130_provenance.
• NP772503.RAbXmzitMMjLaBdAhRjFMKRzF4z3YcZoxyallfBh_O81M130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772503.RAbXmzitMMjLaBdAhRjFMKRzF4z3YcZoxyallfBh_O81M130_provenance.
• assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP606134.RADWUklTnprcXFFrjFuazf1Xe36jBRQ9Zbt7QWwQjchmc130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP606134.RADWUklTnprcXFFrjFuazf1Xe36jBRQ9Zbt7QWwQjchmc130_provenance.
• NP873295.RAvUyfJu9xmwmYqXUe-SzzPyOdfPEhvZJKV_JqvpU09e8130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP873295.RAvUyfJu9xmwmYqXUe-SzzPyOdfPEhvZJKV_JqvpU09e8130_provenance.
• NP914177.RAlTZbTGHHKV8Bnu9UqLluM2GRmeLdLvvvq2hIyJdFcAY130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP914177.RAlTZbTGHHKV8Bnu9UqLluM2GRmeLdLvvvq2hIyJdFcAY130_provenance.
• NP772498.RAh0XJlwUXJ52zDv_TTccfRfN7ui0DCh_VF_qtSNmkkUs130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772498.RAh0XJlwUXJ52zDv_TTccfRfN7ui0DCh_VF_qtSNmkkUs130_provenance.
• NP772499.RAirl2U7q_RQGqhS8iCHJz3en1WmpEdTsASXJiK2eXZsg130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772499.RAirl2U7q_RQGqhS8iCHJz3en1WmpEdTsASXJiK2eXZsg130_provenance.
• NP772490.RAOPKKQhUMFdH1I1_Ev2OHSTcG7kpAZP-cwsFKzfKj_HM130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772490.RAOPKKQhUMFdH1I1_Ev2OHSTcG7kpAZP-cwsFKzfKj_HM130_provenance.
• NP772493.RAPWIaElmk0FuOG2ny9eR_o4cFglpu68Mrrbsu2ko3RU0130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772493.RAPWIaElmk0FuOG2ny9eR_o4cFglpu68Mrrbsu2ko3RU0130_provenance.
• NP772502.RAJbPoScnPg6-w5Tqqn18BZy1uaNIinCExaJRY-K6Zm3Y130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772502.RAJbPoScnPg6-w5Tqqn18BZy1uaNIinCExaJRY-K6Zm3Y130_provenance.
• NP978645.RAK2Ti-NhAYjnZGS6BbQTz3DgrPrR_BbOvevKya67d_hI130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP978645.RAK2Ti-NhAYjnZGS6BbQTz3DgrPrR_BbOvevKya67d_hI130_provenance.
• NP772497.RAyuMTgQCi5TRKD-wunZ2ZKJI1dOiaX5-ESBL5hmJPU8M130_assertion description "[In patients studied for concurrent methylation of several promoters, t-MDS/AML were significantly more frequently hypermethylated in 2 or more promoter regions than de novo MDS or AML suggesting that promoter hypermethylation of genes involved in cell cycle control, apoptosis and DNA repair pathways is a frequent finding in t-MDS/AML and may contribute to secondary leukemogenesis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP772497.RAyuMTgQCi5TRKD-wunZ2ZKJI1dOiaX5-ESBL5hmJPU8M130_provenance.