Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP621196.RAn6rig8y-NLeBkCVVf0J7MW72WYrnYpMY2_fSZlNeK1A130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP621196.RAn6rig8y-NLeBkCVVf0J7MW72WYrnYpMY2_fSZlNeK1A130_provenance.
• NP852548.RAa2RBJ063dmdriG_mmpD2I6nUzaCFm5X3yLA7TX8i7PU130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP852548.RAa2RBJ063dmdriG_mmpD2I6nUzaCFm5X3yLA7TX8i7PU130_provenance.
• assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP1032783.RAVtZ0WKqLObrkCaocaridvpeZ-Hvw2shFtHCeWom1MM0130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1032783.RAVtZ0WKqLObrkCaocaridvpeZ-Hvw2shFtHCeWom1MM0130_provenance.
• NP909108.RAl2LVKldmiSbhI4o0s-iUUVWcjXD9IIoiew6R5HOOdEI130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP909108.RAl2LVKldmiSbhI4o0s-iUUVWcjXD9IIoiew6R5HOOdEI130_provenance.
• NP1032779.RAs-gAm_EOdxmkHD2WBRSoB9hxExOhTy3Qg1yFpgO0izM130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1032779.RAs-gAm_EOdxmkHD2WBRSoB9hxExOhTy3Qg1yFpgO0izM130_provenance.
• NP1032784.RAkLRDOPRYCgm65bhylXWx1Moh81vzxftzSj9tIeGBOHU130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1032784.RAkLRDOPRYCgm65bhylXWx1Moh81vzxftzSj9tIeGBOHU130_provenance.
• NP1032785.RAr9_FfzbeYCoGaUaLb6iulqwQkyK3rbYrHs5u6WM8GDk130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1032785.RAr9_FfzbeYCoGaUaLb6iulqwQkyK3rbYrHs5u6WM8GDk130_provenance.
• NP909012.RANFbvjetbb8GNR4SVf5FQFe4j0yix6awMYcTtysA_Dy4130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP909012.RANFbvjetbb8GNR4SVf5FQFe4j0yix6awMYcTtysA_Dy4130_provenance.
• NP1032782.RAJFvJVBL0nRGnquCVkf8In6baylWitvHIhWAV4Pr2Hv4130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1032782.RAJFvJVBL0nRGnquCVkf8In6baylWitvHIhWAV4Pr2Hv4130_provenance.
• NP897860.RAzlyPXKBXr9bEXZ3xFTTQ4QpHXffoFg7_UtiOV9RdodI130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP897860.RAzlyPXKBXr9bEXZ3xFTTQ4QpHXffoFg7_UtiOV9RdodI130_provenance.
• NP897978.RA5ubLsGGsGAMMx5xN01t2uQ70P-YbEPdrQa8A5bylXMU130_assertion description "[In the absence of IL-17 signaling, IL-22 was the main contributor to skin inflammation, which provides a molecular mechanism for the association of Act1(D10N) with psoriasis susceptibility.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP897978.RA5ubLsGGsGAMMx5xN01t2uQ70P-YbEPdrQa8A5bylXMU130_provenance.