Matches in Nanopublications for { ?s ?p "[In this issue of Science Signaling, Umapathy et al. identify the kinase extracellular signal-regulated kinase 5 (ERK5) as a central mediator that enables ALK to boost MYCN expression, and they show that inhibiting ERK5 in concert with ALK reduced neuroblastoma cell viability in vitro and in xenograft tumor models.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- assertion description "[In this issue of Science Signaling, Umapathy et al. identify the kinase extracellular signal-regulated kinase 5 (ERK5) as a central mediator that enables ALK to boost MYCN expression, and they show that inhibiting ERK5 in concert with ALK reduced neuroblastoma cell viability in vitro and in xenograft tumor models.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[In this issue of Science Signaling, Umapathy et al. identify the kinase extracellular signal-regulated kinase 5 (ERK5) as a central mediator that enables ALK to boost MYCN expression, and they show that inhibiting ERK5 in concert with ALK reduced neuroblastoma cell viability in vitro and in xenograft tumor models.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP1233184.RA3XLy4Xb8EgfkwvGgJ73cwJ3_H_InY8k5EA1vLSksNx8130_assertion description "[In this issue of Science Signaling, Umapathy et al. identify the kinase extracellular signal-regulated kinase 5 (ERK5) as a central mediator that enables ALK to boost MYCN expression, and they show that inhibiting ERK5 in concert with ALK reduced neuroblastoma cell viability in vitro and in xenograft tumor models.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1233184.RA3XLy4Xb8EgfkwvGgJ73cwJ3_H_InY8k5EA1vLSksNx8130_provenance.
- NP1233193.RAzBnFxvIFjZAepHu1q7NekpqlEOCvkl_NCwobbW5-gMQ130_assertion description "[In this issue of Science Signaling, Umapathy et al. identify the kinase extracellular signal-regulated kinase 5 (ERK5) as a central mediator that enables ALK to boost MYCN expression, and they show that inhibiting ERK5 in concert with ALK reduced neuroblastoma cell viability in vitro and in xenograft tumor models.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1233193.RAzBnFxvIFjZAepHu1q7NekpqlEOCvkl_NCwobbW5-gMQ130_provenance.