Matches in Nanopublications for { ?s ?p "[Nonobese diabetic (NOD) mice spontaneously develop diabetes as a consequence of an autoimmune process that can be inhibited by immunotherapy with the 60-kDa heat shock protein (hsp60), with its mycobacterial counterpart 65-kDa (hsp65), or with other Ags such as insulin and glutamic acid decarboxylase (GAD).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP373797.RAeKQdK6_4mmI0A4SVey85M6PR4oDA7Cy7FiXM1odwhag130_assertion description "[Nonobese diabetic (NOD) mice spontaneously develop diabetes as a consequence of an autoimmune process that can be inhibited by immunotherapy with the 60-kDa heat shock protein (hsp60), with its mycobacterial counterpart 65-kDa (hsp65), or with other Ags such as insulin and glutamic acid decarboxylase (GAD).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP373797.RAeKQdK6_4mmI0A4SVey85M6PR4oDA7Cy7FiXM1odwhag130_provenance.
- NP373804.RAZ5WhF9k2NND3cgAQi4bjilRCr4Ul23BerOAG6Y7R0kw130_assertion description "[Nonobese diabetic (NOD) mice spontaneously develop diabetes as a consequence of an autoimmune process that can be inhibited by immunotherapy with the 60-kDa heat shock protein (hsp60), with its mycobacterial counterpart 65-kDa (hsp65), or with other Ags such as insulin and glutamic acid decarboxylase (GAD).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP373804.RAZ5WhF9k2NND3cgAQi4bjilRCr4Ul23BerOAG6Y7R0kw130_provenance.