Matches in Nanopublications for { ?s ?p "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP592298.RAZJefSbuQF8Wj94wismXfWWI14-Ayuzqph-gZf7bfsyE130_assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP592298.RAZJefSbuQF8Wj94wismXfWWI14-Ayuzqph-gZf7bfsyE130_provenance.
- assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP1120119.RAZNOWP7thCsHkhfz18xdTx4KCrVQCAMiduY7Bgu7DaFk130_assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1120119.RAZNOWP7thCsHkhfz18xdTx4KCrVQCAMiduY7Bgu7DaFk130_provenance.
- NP1120120.RATyEb4hmbxVgR5NWD6dApD9pS6BftgACi4NI23e4HEmY130_assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1120120.RATyEb4hmbxVgR5NWD6dApD9pS6BftgACi4NI23e4HEmY130_provenance.
- NP770262.RAAjgWqqaJhHJOo-VAAcd14aPGcpKsDf3Nc4MEhKhEO_4130_assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP770262.RAAjgWqqaJhHJOo-VAAcd14aPGcpKsDf3Nc4MEhKhEO_4130_provenance.
- NP1120117.RAo5NLES9MZbZZd5WjlVvOru88CL3tGIeWc9lHwxray1c130_assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1120117.RAo5NLES9MZbZZd5WjlVvOru88CL3tGIeWc9lHwxray1c130_provenance.
- NP1120118.RAjZorzczXswZ3KhezjAkuKZyM07DF6w_YNBO_YB6iftg130_assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1120118.RAjZorzczXswZ3KhezjAkuKZyM07DF6w_YNBO_YB6iftg130_provenance.
- NP500993.RA2PtmH1N6cHsXLA07TDHw-XLb1_Ti5W2sErFVpB4CWEw130_assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP500993.RA2PtmH1N6cHsXLA07TDHw-XLb1_Ti5W2sErFVpB4CWEw130_provenance.
- NP672321.RALuCwVp5OQYF2eYlix5l-_owRSiIZ9T7qH-QDQI5HvT0130_assertion description "[Our current results suggest that the abnormal lower expression of NME1 in ESCs secrete more IL-8 and VEGF through activation of MAPK/ERK1/2 and AKT signal pathways, up-regulate the level of CD62E and CD105, and finally lead to numerous angiogenesis of vascular endothelial cells in the endometriotic milieu, which is beneficial to the origin and development of endometriosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP672321.RALuCwVp5OQYF2eYlix5l-_owRSiIZ9T7qH-QDQI5HvT0130_provenance.