Matches in Nanopublications for { ?s ?p "[PU.1 downregulation within hematopoietic stem and progenitor cells (HSPCs) is the primary mechanism for the development of acute myeloid leukemia (AML) in mice with homozygous deletion of the upstream regulatory element (URE) of PU.1 gene.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP710248.RAdxZ9qyBROYtzlNTfZ2IEncglGnL_poKaH15CKoihog0130_assertion description "[PU.1 downregulation within hematopoietic stem and progenitor cells (HSPCs) is the primary mechanism for the development of acute myeloid leukemia (AML) in mice with homozygous deletion of the upstream regulatory element (URE) of PU.1 gene.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP710248.RAdxZ9qyBROYtzlNTfZ2IEncglGnL_poKaH15CKoihog0130_provenance.
- NP1118636.RA0iJQWUa-5iMwup4rWKhRpJQvvEB7i9xYhgQsHilcfjY130_assertion description "[PU.1 downregulation within hematopoietic stem and progenitor cells (HSPCs) is the primary mechanism for the development of acute myeloid leukemia (AML) in mice with homozygous deletion of the upstream regulatory element (URE) of PU.1 gene.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1118636.RA0iJQWUa-5iMwup4rWKhRpJQvvEB7i9xYhgQsHilcfjY130_provenance.