Matches in Nanopublications for { ?s ?p "[Similarly, human HS showed genetic or epigenetic inactivation of PTEN, p16(INK4A), and p14(ARF), supporting the relevance of this genetically engineered mouse model of HS.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP199995.RAdOKcUdCHC3yJsELIdGr_vhOmbArGr1w5oh1TApR6If8130_assertion description "[Similarly, human HS showed genetic or epigenetic inactivation of PTEN, p16(INK4A), and p14(ARF), supporting the relevance of this genetically engineered mouse model of HS.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP199995.RAdOKcUdCHC3yJsELIdGr_vhOmbArGr1w5oh1TApR6If8130_provenance.
- NP365710.RA_8CbnA4DJSYh_2N1iB3PVDBu6dffOv5r2U29clApQzQ130_assertion description "[Similarly, human HS showed genetic or epigenetic inactivation of PTEN, p16(INK4A), and p14(ARF), supporting the relevance of this genetically engineered mouse model of HS.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP365710.RA_8CbnA4DJSYh_2N1iB3PVDBu6dffOv5r2U29clApQzQ130_provenance.
- NP282355.RAhxlMhhLBVcHJ_RDljkxFDUBDrKdmVoSCzAHrvBYplTU130_assertion description "[Similarly, human HS showed genetic or epigenetic inactivation of PTEN, p16(INK4A), and p14(ARF), supporting the relevance of this genetically engineered mouse model of HS.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP282355.RAhxlMhhLBVcHJ_RDljkxFDUBDrKdmVoSCzAHrvBYplTU130_provenance.
- NP891005.RAZWMIGJ-7Rc1e_sT8rlGqQA7ypAy_xV21ltkQH6sIC3o130_assertion description "[Similarly, human HS showed genetic or epigenetic inactivation of PTEN, p16(INK4A), and p14(ARF), supporting the relevance of this genetically engineered mouse model of HS.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP891005.RAZWMIGJ-7Rc1e_sT8rlGqQA7ypAy_xV21ltkQH6sIC3o130_provenance.
- NP250155.RAEn_jp5OhcPBB4ulqeKSf0glN1gUSLmeQD1svCtl66ls130_assertion description "[Similarly, human HS showed genetic or epigenetic inactivation of PTEN, p16(INK4A), and p14(ARF), supporting the relevance of this genetically engineered mouse model of HS.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP250155.RAEn_jp5OhcPBB4ulqeKSf0glN1gUSLmeQD1svCtl66ls130_provenance.
- NP571202.RA0orecYcA4cSbKpw6g4Ym7H9jx-6VqQfttHgdJ7glq_M130_assertion description "[Similarly, human HS showed genetic or epigenetic inactivation of PTEN, p16(INK4A), and p14(ARF), supporting the relevance of this genetically engineered mouse model of HS.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP571202.RA0orecYcA4cSbKpw6g4Ym7H9jx-6VqQfttHgdJ7glq_M130_provenance.
- NP167028.RAJB6_Y3Uag5jwcJT61gTmk1c4xgEDYgE4pU_RVH61_Kg130_assertion description "[Similarly, human HS showed genetic or epigenetic inactivation of PTEN, p16(INK4A), and p14(ARF), supporting the relevance of this genetically engineered mouse model of HS.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP167028.RAJB6_Y3Uag5jwcJT61gTmk1c4xgEDYgE4pU_RVH61_Kg130_provenance.