Matches in Nanopublications for { ?s ?p "[Since DTctGMCSF enters and kills its target cells by unique mechanisms (GMCSF-receptor binding and protein synthesis inhibition) and is not similar in structure to Pgp or MRP substrates, we postulated that it would be an active agent against therapy-resistant AML.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP169853.RAQVE_b7ZQVVzD5OUvcThcT2uOIjCMxuP1lNTDXYQZl8M130_assertion description "[Since DTctGMCSF enters and kills its target cells by unique mechanisms (GMCSF-receptor binding and protein synthesis inhibition) and is not similar in structure to Pgp or MRP substrates, we postulated that it would be an active agent against therapy-resistant AML.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP169853.RAQVE_b7ZQVVzD5OUvcThcT2uOIjCMxuP1lNTDXYQZl8M130_provenance.
- NP1373829.RAziOr8NWkE94ARBNxRHUSAp5_WEJUSCFrn-INtnvdJbQ130_assertion description "[Since DTctGMCSF enters and kills its target cells by unique mechanisms (GMCSF-receptor binding and protein synthesis inhibition) and is not similar in structure to Pgp or MRP substrates, we postulated that it would be an active agent against therapy-resistant AML.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1373829.RAziOr8NWkE94ARBNxRHUSAp5_WEJUSCFrn-INtnvdJbQ130_provenance.