Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP376947.RAjA4aF6zAEJmR8aM4pD_KWQ812dz1a0ufvaBJNE5ijo0130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP376947.RAjA4aF6zAEJmR8aM4pD_KWQ812dz1a0ufvaBJNE5ijo0130_provenance.
• assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP872475.RAYxhC7dg7PNsaYnmOJeAFGtFiL3MnvILdYx4-fPG0qgw130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP872475.RAYxhC7dg7PNsaYnmOJeAFGtFiL3MnvILdYx4-fPG0qgw130_provenance.
• assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP690079.RAB5-B2znBC4sYnnAPthqrXdKBiLnb1ne96K3yed7HY9c130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP690079.RAB5-B2znBC4sYnnAPthqrXdKBiLnb1ne96K3yed7HY9c130_provenance.
• assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP690077.RAZnEvQfGs5dcVuwYL8aJ1H4Dkk77wd4qEnWJQiSIQMvo130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP690077.RAZnEvQfGs5dcVuwYL8aJ1H4Dkk77wd4qEnWJQiSIQMvo130_provenance.
• NP488334.RAoV1kVRlW3gA1FBakStrlf1tTajIa4WejjBCsxvmvp4M130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP488334.RAoV1kVRlW3gA1FBakStrlf1tTajIa4WejjBCsxvmvp4M130_provenance.
• NP970794.RAtx-_0beP34CyxmdPUp-ZHFB7l6iDNSQcfIct0b7XCAg130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP970794.RAtx-_0beP34CyxmdPUp-ZHFB7l6iDNSQcfIct0b7XCAg130_provenance.
• NP690076.RAlX0NdMvIh3gKuoVBcpTs6epcQywrkvRHilrGMiFAsPY130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP690076.RAlX0NdMvIh3gKuoVBcpTs6epcQywrkvRHilrGMiFAsPY130_provenance.
• NP690078.RAvbGDn0oBd2C9lwtVuJO48MIWfTHOZWsRhwiqEUGAdS0130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP690078.RAvbGDn0oBd2C9lwtVuJO48MIWfTHOZWsRhwiqEUGAdS0130_provenance.
• NP373287.RA2JACVUfgLtZItr7UWqMONWaCQeiXpdbwceec3w8ay00130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP373287.RA2JACVUfgLtZItr7UWqMONWaCQeiXpdbwceec3w8ay00130_provenance.
• NP490367.RA6iOezVtJc6Cge2Ku91Ge3WvNFYbl8IFZhGMGkY77Xck130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP490367.RA6iOezVtJc6Cge2Ku91Ge3WvNFYbl8IFZhGMGkY77Xck130_provenance.
• NP690075.RAHaHvmXPF-PmZ40z9rGv5KVQVLh9JHgYYoGxEQ0WlA5g130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP690075.RAHaHvmXPF-PmZ40z9rGv5KVQVLh9JHgYYoGxEQ0WlA5g130_provenance.
• NP690074.RAzZ_j7R42s6Hhv-23fgiAbPw5X-W3QPzHamM-QLhvMWg130_assertion description "[Since IL-1 and TNF-alpha are related to inflammation and their immunoexpression increases in PC, inhibition of these cytokines might be a possible target for PC treatment, because they decrease the activity of all transduction pathway members that activate transcription factors such as NF-kappaB, Elk-1 or ATF-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP690074.RAzZ_j7R42s6Hhv-23fgiAbPw5X-W3QPzHamM-QLhvMWg130_provenance.