Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP55024.RA2saFndTGKnRkNbLDD5uy5iR3tJR0MPBX7DDt05nOhxg130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP55024.RA2saFndTGKnRkNbLDD5uy5iR3tJR0MPBX7DDt05nOhxg130_provenance.
• NP51454.RAFUsX402BK1D9bWR_pnR1uW22TBzosz_oW8HUN_GpJeA130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP51454.RAFUsX402BK1D9bWR_pnR1uW22TBzosz_oW8HUN_GpJeA130_provenance.
• NP702699.RAt6C9nqQ6KGTS3G5ufDWhck07ChPSKczLk1X-D2LlfCE130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP702699.RAt6C9nqQ6KGTS3G5ufDWhck07ChPSKczLk1X-D2LlfCE130_provenance.
• NP69223.RAUSMNAQ-GjMjJeRAMbEYsAqLIOkitKdmX45w_3sf9eTY130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP69223.RAUSMNAQ-GjMjJeRAMbEYsAqLIOkitKdmX45w_3sf9eTY130_provenance.
• assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP63975.RAqvk06biqFhLXRPWi5YbwNYCrcZpWgMRO1cM-HsoSYUI130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP63975.RAqvk06biqFhLXRPWi5YbwNYCrcZpWgMRO1cM-HsoSYUI130_provenance.
• assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP665292.RAfra_V2u1CJRETdRLwvCnciP-IbbYM7mMRPWa5wUz8FE130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP665292.RAfra_V2u1CJRETdRLwvCnciP-IbbYM7mMRPWa5wUz8FE130_provenance.
• NP121557.RAkar8JxHiM4K4--EZVQTA6FR2YFlI2z31PgZgu-uuiHo130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP121557.RAkar8JxHiM4K4--EZVQTA6FR2YFlI2z31PgZgu-uuiHo130_provenance.
• NP121559.RAqGA1dbsoMg4LnneroVouX5wqvwYD139841ERkd72j3g130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP121559.RAqGA1dbsoMg4LnneroVouX5wqvwYD139841ERkd72j3g130_provenance.
• NP86270.RA_OH0jyxpIuq19BGPwJ1OFA_4x1NNZSFIqSc2xDfpyEk130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP86270.RA_OH0jyxpIuq19BGPwJ1OFA_4x1NNZSFIqSc2xDfpyEk130_provenance.
• NP121556.RAIMNmj2LPVLMas9G1yX_Ht3sJstByqNZD_CfBJo_hnU0130_assertion description "[Suggested mechanisms for hyperserotonemia in autism have been increased synthesis of serotonin (5HT) by tryptophan hydroxylase (TPH), increased uptake into platelets through 5HT transporter (5HTt), diminished release from platelets through 5HT2A receptor (5HT2Ar) and decreased metabolism by monoamine oxydase (MAOA).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP121556.RAIMNmj2LPVLMas9G1yX_Ht3sJstByqNZD_CfBJo_hnU0130_provenance.