Matches in Nanopublications for { ?s ?p "[The absence of TLR2, MyD88, and MyD88xTrif conferred both physiologic and histologic protection against sublethal ischemia at 24 h. Interestingly, TLR2-deficient mice were better protected from ischemic renal injury than those deficient for the adapter protein MyD88, raising the intriguing possibility that TLR-2-dependent/MyD88-independent pathways also contribute to kidney injury.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP893098.RArc9t7eChL0ZWKYwPVjX1Y0wxc2qThvPrd9CeUtucgvM130_assertion description "[The absence of TLR2, MyD88, and MyD88xTrif conferred both physiologic and histologic protection against sublethal ischemia at 24 h. Interestingly, TLR2-deficient mice were better protected from ischemic renal injury than those deficient for the adapter protein MyD88, raising the intriguing possibility that TLR-2-dependent/MyD88-independent pathways also contribute to kidney injury.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP893098.RArc9t7eChL0ZWKYwPVjX1Y0wxc2qThvPrd9CeUtucgvM130_provenance.
- NP709797.RAJPZE6OSVRnwSMabOkQ-I-ySIdyRKNNlVu-dKPjQOcSc130_assertion description "[The absence of TLR2, MyD88, and MyD88xTrif conferred both physiologic and histologic protection against sublethal ischemia at 24 h. Interestingly, TLR2-deficient mice were better protected from ischemic renal injury than those deficient for the adapter protein MyD88, raising the intriguing possibility that TLR-2-dependent/MyD88-independent pathways also contribute to kidney injury.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP709797.RAJPZE6OSVRnwSMabOkQ-I-ySIdyRKNNlVu-dKPjQOcSc130_provenance.