Matches in Nanopublications for { ?s ?p "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP237992.RAb1hxKVkegHk-3XY4Mn0HD71_XM8ewIU_S-lldyuwXXg130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP237992.RAb1hxKVkegHk-3XY4Mn0HD71_XM8ewIU_S-lldyuwXXg130_provenance.
- NP669191.RAob1KWP_XgXtDZ41RmzKjBUCCM_cEEH4DpfEsk6xpWrM130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP669191.RAob1KWP_XgXtDZ41RmzKjBUCCM_cEEH4DpfEsk6xpWrM130_provenance.
- NP863080.RAgkTg7mzNkZVPaYCu0aGJhuHxVt2EGPmpKau_DX3FcEM130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP863080.RAgkTg7mzNkZVPaYCu0aGJhuHxVt2EGPmpKau_DX3FcEM130_provenance.
- NP804360.RA3kmjQamGCjahCtRoJTjy1EwSStqHz-ipYc59B7pJ_Z8130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP804360.RA3kmjQamGCjahCtRoJTjy1EwSStqHz-ipYc59B7pJ_Z8130_provenance.
- NP938888.RA0fdY7Gl3kCXVt05zz2uCQ7v1M5E62I72qMxzpj8HukA130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP938888.RA0fdY7Gl3kCXVt05zz2uCQ7v1M5E62I72qMxzpj8HukA130_provenance.
- NP747952.RATyjvPcqCiQHXDEgUe_0KWw2ic4ZVcCH6PypIvqVQN_Y130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP747952.RATyjvPcqCiQHXDEgUe_0KWw2ic4ZVcCH6PypIvqVQN_Y130_provenance.
- assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP534860.RAT7ufmjM493hZLhRc9vNNatOoqieurBTJ8fEZrcJgEFM130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP534860.RAT7ufmjM493hZLhRc9vNNatOoqieurBTJ8fEZrcJgEFM130_provenance.
- NP683611.RAj6Qdt8Oias306Zp0709McJNtVfyn0wADh-lhaXTddqQ130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP683611.RAj6Qdt8Oias306Zp0709McJNtVfyn0wADh-lhaXTddqQ130_provenance.
- NP683633.RAxibO4p9OfSvlY96WQvQHWWcFeEDjYTS5x_3VjV2WB_o130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP683633.RAxibO4p9OfSvlY96WQvQHWWcFeEDjYTS5x_3VjV2WB_o130_provenance.
- NP534863.RA1MOPwTH_rLMs_bZmm-cM7Wpt1wmi7vTw0nTLG6qWtWw130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP534863.RA1MOPwTH_rLMs_bZmm-cM7Wpt1wmi7vTw0nTLG6qWtWw130_provenance.
- NP534864.RA9hZWlzN84cR0hdXP4k0VdklXku5YMnsOA02haEdWl-c130_assertion description "[The finding that TNFRSF1A, SCNN1B and SCNN1G are clinically relevant modulators of CF disease supports current concepts that the depletion of airway surface liquid and inadequate host inflammatory responses trigger pulmonary disease in CF.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP534864.RA9hZWlzN84cR0hdXP4k0VdklXku5YMnsOA02haEdWl-c130_provenance.