Matches in Nanopublications for { ?s ?p "[The findings indicate that TGFBR2 gene mutations are responsible for sCAD in 3.6% (95% CI 0.0 to 8.4) of cases, have implications in understanding the role of TGF? signalling in the pathogenesis of sCAD and emphasise the importance of considering molecular characterisation of the TGFBR2 gene in these patients, regardless of the presence of clinical features suggestive of hereditary connective tissue disorders.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP298324.RABGEyqwjADNfQ_3XWnMo_jI8NO4Hf0cskVJO8Bzy3wXQ130_assertion description "[The findings indicate that TGFBR2 gene mutations are responsible for sCAD in 3.6% (95% CI 0.0 to 8.4) of cases, have implications in understanding the role of TGF? signalling in the pathogenesis of sCAD and emphasise the importance of considering molecular characterisation of the TGFBR2 gene in these patients, regardless of the presence of clinical features suggestive of hereditary connective tissue disorders.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP298324.RABGEyqwjADNfQ_3XWnMo_jI8NO4Hf0cskVJO8Bzy3wXQ130_provenance.
- NP734290.RAZrGOFyxPSnIDrYeP8v_DYAhywEKdbSX8OrXL9j79IrY130_assertion description "[The findings indicate that TGFBR2 gene mutations are responsible for sCAD in 3.6% (95% CI 0.0 to 8.4) of cases, have implications in understanding the role of TGF? signalling in the pathogenesis of sCAD and emphasise the importance of considering molecular characterisation of the TGFBR2 gene in these patients, regardless of the presence of clinical features suggestive of hereditary connective tissue disorders.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP734290.RAZrGOFyxPSnIDrYeP8v_DYAhywEKdbSX8OrXL9j79IrY130_provenance.
- assertion description "[The findings indicate that TGFBR2 gene mutations are responsible for sCAD in 3.6% (95% CI 0.0 to 8.4) of cases, have implications in understanding the role of TGF? signalling in the pathogenesis of sCAD and emphasise the importance of considering molecular characterisation of the TGFBR2 gene in these patients, regardless of the presence of clinical features suggestive of hereditary connective tissue disorders.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP867975.RA2nY8syAYjUeCuWJVOPC8mvZUPYl7waexhVhPTSy2FZ4130_assertion description "[The findings indicate that TGFBR2 gene mutations are responsible for sCAD in 3.6% (95% CI 0.0 to 8.4) of cases, have implications in understanding the role of TGF? signalling in the pathogenesis of sCAD and emphasise the importance of considering molecular characterisation of the TGFBR2 gene in these patients, regardless of the presence of clinical features suggestive of hereditary connective tissue disorders.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP867975.RA2nY8syAYjUeCuWJVOPC8mvZUPYl7waexhVhPTSy2FZ4130_provenance.