Matches in Nanopublications for { ?s ?p "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP874470.RAsaIdaBZ6QkxbW9JOT63xE2Ay68fH8ZXNsSDlqKkqWGA130_assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP874470.RAsaIdaBZ6QkxbW9JOT63xE2Ay68fH8ZXNsSDlqKkqWGA130_provenance.
- NP892989.RAfd2ZuZh-3UPDzPlDRERmq_0zo_axe9b9zGuJKHm_PgY130_assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP892989.RAfd2ZuZh-3UPDzPlDRERmq_0zo_axe9b9zGuJKHm_PgY130_provenance.
- NP721834.RA14dN6TVWRXC4gq2wpShifggAMORakpA7KoAjc1wALdA130_assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP721834.RA14dN6TVWRXC4gq2wpShifggAMORakpA7KoAjc1wALdA130_provenance.
- NP914513.RA2TsaPjZPu7T1RVAkt-lGsnLDv4fTlaiCer95zKCZ6wA130_assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP914513.RA2TsaPjZPu7T1RVAkt-lGsnLDv4fTlaiCer95zKCZ6wA130_provenance.
- NP757218.RARvWlDfLV2yUBVq4lCwZ3vied1HHwz5L5z9Sz8HU7U64130_assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP757218.RARvWlDfLV2yUBVq4lCwZ3vied1HHwz5L5z9Sz8HU7U64130_provenance.
- NP722367.RARSFccMLgrmNeKbFgyeQgtk0ZfpGAQAWxXp26ebwnJwM130_assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP722367.RARSFccMLgrmNeKbFgyeQgtk0ZfpGAQAWxXp26ebwnJwM130_provenance.
- assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP964950.RAd2-5K79MSElhT7HMg4sNmXi6zlHJ5s5NcPMJ9NNwgsE130_assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP964950.RAd2-5K79MSElhT7HMg4sNmXi6zlHJ5s5NcPMJ9NNwgsE130_provenance.
- NP964951.RA3C8klWuK7FI24tqudR4Sfr7S5Kq7z4U06lXaKrq3Gnw130_assertion description "[The identification of TAK1 as a main biochemical mediator of EMT and fibrosis in mesothelial cells from human peritoneum and the study of signalling pathways induced by its activity may be relevant in the design of new therapies aimed to counteract peritoneal fibrosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP964951.RA3C8klWuK7FI24tqudR4Sfr7S5Kq7z4U06lXaKrq3Gnw130_provenance.