Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP743137.RAeKtihdxW36uyW9yXyhYovcHYwr3Tiqdnw2ZDgFk0Rrk130_assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP743137.RAeKtihdxW36uyW9yXyhYovcHYwr3Tiqdnw2ZDgFk0Rrk130_provenance.
• assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP1228947.RAUC3-dsg3sfatWEIkKbP9Zaz-0QVm_YzZhdsmxMMaI1Y130_assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1228947.RAUC3-dsg3sfatWEIkKbP9Zaz-0QVm_YzZhdsmxMMaI1Y130_provenance.
• NP1228948.RAe0MxD4VmYcRqjw3pvU3HlyY0kepK-XcAH4etLdKGN4I130_assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1228948.RAe0MxD4VmYcRqjw3pvU3HlyY0kepK-XcAH4etLdKGN4I130_provenance.
• NP1228953.RASYr7jviJbAYKDI5CUWd-m6-j97LG6-45W4ZVYTFpjtQ130_assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1228953.RASYr7jviJbAYKDI5CUWd-m6-j97LG6-45W4ZVYTFpjtQ130_provenance.
• NP695284.RAt4yVbMQSRTz72cdRMHjaw2oEKqpPNwBzA1o8o9OLDOQ130_assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP695284.RAt4yVbMQSRTz72cdRMHjaw2oEKqpPNwBzA1o8o9OLDOQ130_provenance.
• NP1228950.RAuP2FM0q00MEhBb4kKtRCCqEwS9hSyvPWeVkEjnPYFuw130_assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1228950.RAuP2FM0q00MEhBb4kKtRCCqEwS9hSyvPWeVkEjnPYFuw130_provenance.
• NP488682.RA6its0vYS6MOVz3crnbY_ixFazb9TFKZxylH4n82vu6k130_assertion description "[The peptide nearly totally suppressed the expression of tumor necrosis factor-? and repressed the production of the pro-inflammatory mediators interleukin-1?, nitric oxide and of the transcription factor nuclear factor kappa B. Inhibition of tumor necrosis factor-? likely accounted for the observed down-regulation of nuclear factor kappa B that modulates the transcription of genes specifically involved in amyotrophic lateral sclerosis (sod1 and the glutamate transporter slc1a2).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP488682.RA6its0vYS6MOVz3crnbY_ixFazb9TFKZxylH4n82vu6k130_provenance.