Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP882693.RAeHAXtvP-QqywDIevuH2ZOfoqR0DSCmL64vPOY368-54130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP882693.RAeHAXtvP-QqywDIevuH2ZOfoqR0DSCmL64vPOY368-54130_provenance.
• assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP299165.RAdbC7SKMpigA5UKHjOGL7RuHOcwh6ct295-ZReIF2488130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP299165.RAdbC7SKMpigA5UKHjOGL7RuHOcwh6ct295-ZReIF2488130_provenance.
• assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP915049.RAAAklkJJJiLYIIhlACAb4hijHtBnJBC4ZOgbs0jZxRM4130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP915049.RAAAklkJJJiLYIIhlACAb4hijHtBnJBC4ZOgbs0jZxRM4130_provenance.
• assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP663109.RACcSi0mdxscvZLQVYdKhYTHafjwcgj42_41Rw-UN_pKY130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP663109.RACcSi0mdxscvZLQVYdKhYTHafjwcgj42_41Rw-UN_pKY130_provenance.
• NP915052.RAfRqYGaaMvNyXUvKbS-y-JtIIORiOysFuVQ5YaEizkvs130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP915052.RAfRqYGaaMvNyXUvKbS-y-JtIIORiOysFuVQ5YaEizkvs130_provenance.
• NP931062.RANs5YR3HdKq8qshBtl0qgqweP0RjsofuKtd4bsSqWEfI130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP931062.RANs5YR3HdKq8qshBtl0qgqweP0RjsofuKtd4bsSqWEfI130_provenance.
• NP672662.RA4vBI9azCc551i8F4VlW8bRbgzFwwFqdwofphtQG_ZGo130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP672662.RA4vBI9azCc551i8F4VlW8bRbgzFwwFqdwofphtQG_ZGo130_provenance.
• NP915051.RA1WRRKUldhPn_nPPio3KhG-EIY8S3LF91T3OtNs3u7ro130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP915051.RA1WRRKUldhPn_nPPio3KhG-EIY8S3LF91T3OtNs3u7ro130_provenance.
• NP915053.RAzYEXXyVR39wq5Vomjcs7Efc0PrO9ht4EZgG_agcwma8130_assertion description "[These findings, combined with a Dock6 expression profile that is consistent with an AOS phenotype as well as the very recent demonstration of dominant mutations of ARHGAP31 in AOS, establish Cdc42 and Rac1 as key molecules in the pathogenesis of AOS and suggest that other regulators of these Rho GTPase proteins might be good candidates in the quest to define the genetic spectrum of this genetically heterogeneous condition.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP915053.RAzYEXXyVR39wq5Vomjcs7Efc0PrO9ht4EZgG_agcwma8130_provenance.