Matches in Nanopublications for { ?s ?p "[These results confirm ATR, MED1, and CHK1 as targets of the mutator pathway in stomach tumorigenesis, and also suggest a potential role of MED1 increasing, together with hMSH3 and hMSH6, the genomic instability in the mutator pathway as a secondary mutator.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP335102.RARpdaO24uPSv7C645Pgs5g6HdSd6N6rsu7afWP4iUjYI130_assertion description "[These results confirm ATR, MED1, and CHK1 as targets of the mutator pathway in stomach tumorigenesis, and also suggest a potential role of MED1 increasing, together with hMSH3 and hMSH6, the genomic instability in the mutator pathway as a secondary mutator.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP335102.RARpdaO24uPSv7C645Pgs5g6HdSd6N6rsu7afWP4iUjYI130_provenance.
- NP638176.RAlRl8E3mBSJ8R329L8MyS0vx7hYWK9LPTFdP8adt0eIY130_assertion description "[These results confirm ATR, MED1, and CHK1 as targets of the mutator pathway in stomach tumorigenesis, and also suggest a potential role of MED1 increasing, together with hMSH3 and hMSH6, the genomic instability in the mutator pathway as a secondary mutator.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP638176.RAlRl8E3mBSJ8R329L8MyS0vx7hYWK9LPTFdP8adt0eIY130_provenance.
- NP811644.RAhLq99TCM52COcdiS2Mhz0a37BQy_mD55_m2esE6xwzU130_assertion description "[These results confirm ATR, MED1, and CHK1 as targets of the mutator pathway in stomach tumorigenesis, and also suggest a potential role of MED1 increasing, together with hMSH3 and hMSH6, the genomic instability in the mutator pathway as a secondary mutator.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP811644.RAhLq99TCM52COcdiS2Mhz0a37BQy_mD55_m2esE6xwzU130_provenance.
- NP335107.RAq4fOYzxki6eLa0CluzSx4GcmqNVpSX-UJRgLnFFFtzI130_assertion description "[These results confirm ATR, MED1, and CHK1 as targets of the mutator pathway in stomach tumorigenesis, and also suggest a potential role of MED1 increasing, together with hMSH3 and hMSH6, the genomic instability in the mutator pathway as a secondary mutator.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP335107.RAq4fOYzxki6eLa0CluzSx4GcmqNVpSX-UJRgLnFFFtzI130_provenance.