Matches in Nanopublications for { ?s ?p "[These results suggest that TGFBR2 mutations in primary colon cancers may be responsible for the increased proliferation and cdk4 expression in these tumors and provide evidence that deregulation of cdk4 is a pathogenic in vivo consequence of TGFBR2 inactivation in primary colon cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP573490.RAfj4bSU_HgCd-WjQLWUa50gsvM9mbucE_q067Is-Rqf8130_assertion description "[These results suggest that TGFBR2 mutations in primary colon cancers may be responsible for the increased proliferation and cdk4 expression in these tumors and provide evidence that deregulation of cdk4 is a pathogenic in vivo consequence of TGFBR2 inactivation in primary colon cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP573490.RAfj4bSU_HgCd-WjQLWUa50gsvM9mbucE_q067Is-Rqf8130_provenance.
- assertion description "[These results suggest that TGFBR2 mutations in primary colon cancers may be responsible for the increased proliferation and cdk4 expression in these tumors and provide evidence that deregulation of cdk4 is a pathogenic in vivo consequence of TGFBR2 inactivation in primary colon cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP301580.RARetERMXkD2lp3QI2QevVNQdnJ0NUbvaxKtNIAFXmAvg130_assertion description "[These results suggest that TGFBR2 mutations in primary colon cancers may be responsible for the increased proliferation and cdk4 expression in these tumors and provide evidence that deregulation of cdk4 is a pathogenic in vivo consequence of TGFBR2 inactivation in primary colon cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP301580.RARetERMXkD2lp3QI2QevVNQdnJ0NUbvaxKtNIAFXmAvg130_provenance.
- assertion description "[These results suggest that TGFBR2 mutations in primary colon cancers may be responsible for the increased proliferation and cdk4 expression in these tumors and provide evidence that deregulation of cdk4 is a pathogenic in vivo consequence of TGFBR2 inactivation in primary colon cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP734518.RArQtf9qPIRyDIlyhxHOhONJkvb1FrMb8I0oTvuSweyeU130_assertion description "[These results suggest that TGFBR2 mutations in primary colon cancers may be responsible for the increased proliferation and cdk4 expression in these tumors and provide evidence that deregulation of cdk4 is a pathogenic in vivo consequence of TGFBR2 inactivation in primary colon cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP734518.RArQtf9qPIRyDIlyhxHOhONJkvb1FrMb8I0oTvuSweyeU130_provenance.
- NP509515.RApOPVs2cPZoL0crsCxy-th-8OfW6KC2UB2x7Bc25A524130_assertion description "[These results suggest that TGFBR2 mutations in primary colon cancers may be responsible for the increased proliferation and cdk4 expression in these tumors and provide evidence that deregulation of cdk4 is a pathogenic in vivo consequence of TGFBR2 inactivation in primary colon cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP509515.RApOPVs2cPZoL0crsCxy-th-8OfW6KC2UB2x7Bc25A524130_provenance.
- NP509514.RAOAudJm-POvG_xHT4nYcI9nZDrFhpbwnvg5V6PgDzcx0130_assertion description "[These results suggest that TGFBR2 mutations in primary colon cancers may be responsible for the increased proliferation and cdk4 expression in these tumors and provide evidence that deregulation of cdk4 is a pathogenic in vivo consequence of TGFBR2 inactivation in primary colon cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP509514.RAOAudJm-POvG_xHT4nYcI9nZDrFhpbwnvg5V6PgDzcx0130_provenance.