Matches in Nanopublications for { ?s ?p "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
Showing items 1 to 8 of
8
with 100 items per page.
- NP852644.RAaPc8F9hpK0K7nXafDv4dVXohyVmwS4AmfnaTA4Cx5lA130_assertion description "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP852644.RAaPc8F9hpK0K7nXafDv4dVXohyVmwS4AmfnaTA4Cx5lA130_provenance.
- NP865279.RAxtwXDqzfP_EtZxFzZVC-LBUMaiiFoFw89vyxFauHD-A130_assertion description "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP865279.RAxtwXDqzfP_EtZxFzZVC-LBUMaiiFoFw89vyxFauHD-A130_provenance.
- assertion description "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP949127.RAqnKq1K2eXODygwtGMbIYp8Enmt6frR_QLhmD4YlMmCo130_assertion description "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP949127.RAqnKq1K2eXODygwtGMbIYp8Enmt6frR_QLhmD4YlMmCo130_provenance.
- NP527372.RA0GH79L6k734y_JXd1A1UxBTQ9HruRhv8g91kq97MRHI130_assertion description "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP527372.RA0GH79L6k734y_JXd1A1UxBTQ9HruRhv8g91kq97MRHI130_provenance.
- NP527508.RAyaKSW507EBIHfIpsUtPwcVKg_E_cJFuv4oHng8JztFs130_assertion description "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP527508.RAyaKSW507EBIHfIpsUtPwcVKg_E_cJFuv4oHng8JztFs130_provenance.
- NP949128.RAGAUTCtkOAGQ0clnBprhthseds_ScNyaSCsL9xTHwqYU130_assertion description "[This review summarizes studies providing evidence (1) that endogenous RAS activation regulates important physiological events during ovulation and luteinization (2) that expression of the mutant, active KRAS(G12D) in granulosa cells in vivo causes abnormal follicle growth arrest leading to premature ovarian failure and (3) that KRAS(G12D) expression in ovarian surface epithelial (OSE) cells renders them susceptible to the pathological outcome of transformation and tumor formation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP949128.RAGAUTCtkOAGQ0clnBprhthseds_ScNyaSCsL9xTHwqYU130_provenance.