Matches in Nanopublications for { ?s ?p "[Together, these data suggest that a previously unrecognized cross-talk mechanism between the STAT3 and PKC?II signaling pathways provides the molecular basis for the tumor-induced blockade in the differentiation of myeloid cells, and suggest that enhancing PKC?II activity may be a therapeutic strategy to alleviate cancer-mediated suppression of the immune system.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP841923.RAYfhbZkiiaHcySHqU7rYgfTvAy7iuKlwycz03s-lf6oY130_assertion description "[Together, these data suggest that a previously unrecognized cross-talk mechanism between the STAT3 and PKC?II signaling pathways provides the molecular basis for the tumor-induced blockade in the differentiation of myeloid cells, and suggest that enhancing PKC?II activity may be a therapeutic strategy to alleviate cancer-mediated suppression of the immune system.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP841923.RAYfhbZkiiaHcySHqU7rYgfTvAy7iuKlwycz03s-lf6oY130_provenance.
- NP842154.RASuizDFcRPcavN4r3M4aSUlUvGuKlcMeo6523B3uhb44130_assertion description "[Together, these data suggest that a previously unrecognized cross-talk mechanism between the STAT3 and PKC?II signaling pathways provides the molecular basis for the tumor-induced blockade in the differentiation of myeloid cells, and suggest that enhancing PKC?II activity may be a therapeutic strategy to alleviate cancer-mediated suppression of the immune system.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP842154.RASuizDFcRPcavN4r3M4aSUlUvGuKlcMeo6523B3uhb44130_provenance.
- NP587612.RAtGrQghwiS2xmgW3-I6WYzuoWtzxckPCxRA2YD6N1P2A130_assertion description "[Together, these data suggest that a previously unrecognized cross-talk mechanism between the STAT3 and PKC?II signaling pathways provides the molecular basis for the tumor-induced blockade in the differentiation of myeloid cells, and suggest that enhancing PKC?II activity may be a therapeutic strategy to alleviate cancer-mediated suppression of the immune system.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP587612.RAtGrQghwiS2xmgW3-I6WYzuoWtzxckPCxRA2YD6N1P2A130_provenance.
- NP587955.RAGvgKv-jMy7Y7LHrTKdtQQChtBDKPNq8ubKEgrKv4_Yc130_assertion description "[Together, these data suggest that a previously unrecognized cross-talk mechanism between the STAT3 and PKC?II signaling pathways provides the molecular basis for the tumor-induced blockade in the differentiation of myeloid cells, and suggest that enhancing PKC?II activity may be a therapeutic strategy to alleviate cancer-mediated suppression of the immune system.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP587955.RAGvgKv-jMy7Y7LHrTKdtQQChtBDKPNq8ubKEgrKv4_Yc130_provenance.