Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP370420.RAe9Qp5wBjN1ssjfVHTgFFKsr84mfFoy1dxq7ukH1jBZo130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP370420.RAe9Qp5wBjN1ssjfVHTgFFKsr84mfFoy1dxq7ukH1jBZo130_provenance.
• NP191849.RAhQj3J2vmNMFxtRrTxhXG911X8hpadFfAk3KFekMDxcQ130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP191849.RAhQj3J2vmNMFxtRrTxhXG911X8hpadFfAk3KFekMDxcQ130_provenance.
• NP716600.RAhf9cTsGsALOQ1-y6vcwRyTKb3LQ0n6AVAPii1uRDrUo130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP716600.RAhf9cTsGsALOQ1-y6vcwRyTKb3LQ0n6AVAPii1uRDrUo130_provenance.
• NP832098.RAs8lfZm2gXFb8L0Foy02u4Vw8IjuSqVGcKt5Nb2ra8oY130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP832098.RAs8lfZm2gXFb8L0Foy02u4Vw8IjuSqVGcKt5Nb2ra8oY130_provenance.
• NP331445.RAHOhzmiIvNzZmuDDx1xKddxvcyiqESg2Dtq1tKdwhRvc130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP331445.RAHOhzmiIvNzZmuDDx1xKddxvcyiqESg2Dtq1tKdwhRvc130_provenance.
• assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP280548.RAcTDCYuQwLTtyyk-7aSBuJpCAfV7G865hOFQ_tNCQoqA130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP280548.RAcTDCYuQwLTtyyk-7aSBuJpCAfV7G865hOFQ_tNCQoqA130_provenance.
• NP453028.RAKI_63bzdIfGW7sPUpqWbQZTcxHaKNyNtBue7m9GqjOE130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP453028.RAKI_63bzdIfGW7sPUpqWbQZTcxHaKNyNtBue7m9GqjOE130_provenance.
• assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP748168.RA_9Ov3vionjinbSriyf0R8kR98U-bdhelAcktPH2QwPA130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP748168.RA_9Ov3vionjinbSriyf0R8kR98U-bdhelAcktPH2QwPA130_provenance.
• NP730238.RA_JLrM4_FQqIYm5fUBgEIolw-RVhZLJzYuNvl6ChBp9w130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP730238.RA_JLrM4_FQqIYm5fUBgEIolw-RVhZLJzYuNvl6ChBp9w130_provenance.
• NP730243.RAyBJPaay0vfb9bBhTyrvg6IzvrSOqkc6ViaQTKZd8hGk130_assertion description "[Unexpectedly, these changes were independent of changes in proliferation rate within the hyperplastic tissue, suggesting that endogenous SST controls GHRH-induced adenoma formation primarily via modulation of apoptotic and/or cellular senescence pathways, consistent with the predicted function of some of the most differentially expressed genes (Casp1, MAP2K1, TNFR2) identified by membrane arrays and confirmed by quantitative real-time RT-PCR.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP730243.RAyBJPaay0vfb9bBhTyrvg6IzvrSOqkc6ViaQTKZd8hGk130_provenance.