Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP801678.RAcJPe05_04m_8g3upOKRqVtsMrkDm3LLd0utEWyAWedA130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP801678.RAcJPe05_04m_8g3upOKRqVtsMrkDm3LLd0utEWyAWedA130_provenance.
• assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP808264.RA9WOIlNcawNVpQXdZOxOW3ebpjSQoRMoj_YyLN1U3-qY130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP808264.RA9WOIlNcawNVpQXdZOxOW3ebpjSQoRMoj_YyLN1U3-qY130_provenance.
• NP660876.RAJLqT8WWeJ4vOc2JGMu-28GbRI-UIGhbEZpCg4fm38Pc130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP660876.RAJLqT8WWeJ4vOc2JGMu-28GbRI-UIGhbEZpCg4fm38Pc130_provenance.
• assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP645090.RAAwmcuF43KzS8kzs8Fps_3ZRxkgQLFNkegMM7sxBfaxM130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP645090.RAAwmcuF43KzS8kzs8Fps_3ZRxkgQLFNkegMM7sxBfaxM130_provenance.
• NP587634.RArBXMa1Ee5NaAeYuvUAMyTc2zxBiYTWzpyEUblxU5QBE130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP587634.RArBXMa1Ee5NaAeYuvUAMyTc2zxBiYTWzpyEUblxU5QBE130_provenance.
• NP587635.RAncfdluqsGgt-OTuNwlnJ0_F7pvkSTuF8hV5mUMyiHPw130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP587635.RAncfdluqsGgt-OTuNwlnJ0_F7pvkSTuF8hV5mUMyiHPw130_provenance.
• NP587637.RAjmROB0BVqnJbKztrAY25g836JH4iFuEO1LXbfbZD4wc130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP587637.RAjmROB0BVqnJbKztrAY25g836JH4iFuEO1LXbfbZD4wc130_provenance.
• NP373302.RAIpNmO_8uUylLM3zvYBT8dBSrgxKGltnFaajbXNn_fpE130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP373302.RAIpNmO_8uUylLM3zvYBT8dBSrgxKGltnFaajbXNn_fpE130_provenance.
• NP970812.RA45WBWFDWh6eubUWCjuY6UPaupS2TInUWkx8xiiD51JQ130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP970812.RA45WBWFDWh6eubUWCjuY6UPaupS2TInUWkx8xiiD51JQ130_provenance.
• NP587641.RAw-IN5-FpZ94xYAvpjQB-6OjyyVpJ3VFnzvEFono80Dk130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP587641.RAw-IN5-FpZ94xYAvpjQB-6OjyyVpJ3VFnzvEFono80Dk130_provenance.
• NP872491.RA9DQI31YHfnX_zmFUbwYdbec8MdaFLXPsz-M5r7L5Wp0130_assertion description "[Using inhibitors and dominant negative constructs, we have demonstrated that RET(Y791F) and RET(S891A) induce STAT3 Ser(727) phosphorylation via a canonical Ras/ERK1/2 pathway and that integration of the Ras/ERK1/2/ELK-1 and STAT3 pathways was required for up-regulation of the c-fos promoter by FMTC-RET.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP872491.RA9DQI31YHfnX_zmFUbwYdbec8MdaFLXPsz-M5r7L5Wp0130_provenance.