Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP563128.RAdA4XCRv3UgS7rBG0WojwH50FAr95IjcrFjVtWGPLmTA130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP563128.RAdA4XCRv3UgS7rBG0WojwH50FAr95IjcrFjVtWGPLmTA130_provenance.
• NP360392.RAZ6wK1rGiUeiKULeawLUCRQibfNuPON01tgCd67hrdao130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP360392.RAZ6wK1rGiUeiKULeawLUCRQibfNuPON01tgCd67hrdao130_provenance.
• NP280657.RAkAQHuSJYx7yYMPcbxC4gcK1gXd5QQcDUJDUj3OiR02Y130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP280657.RAkAQHuSJYx7yYMPcbxC4gcK1gXd5QQcDUJDUj3OiR02Y130_provenance.
• NP666403.RAlErTEyUQRKVx40WRWsmnJRSUuAYZSrN2_YR86dz0XVQ130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP666403.RAlErTEyUQRKVx40WRWsmnJRSUuAYZSrN2_YR86dz0XVQ130_provenance.
• NP610239.RArb82KOHLIPzlj_cazOm8itXcIDuRfTFvfazoAO1I4ec130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP610239.RArb82KOHLIPzlj_cazOm8itXcIDuRfTFvfazoAO1I4ec130_provenance.
• assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP568107.RA4Vx4pcakSl-X6JlMX16BWA9v0vLUSYY2aYHlLjZxOkI130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP568107.RA4Vx4pcakSl-X6JlMX16BWA9v0vLUSYY2aYHlLjZxOkI130_provenance.
• NP400364.RAcSwGKwg1aWGIudrkBj9kd3zdIQPcIFR0MOwcbZMgsLk130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP400364.RAcSwGKwg1aWGIudrkBj9kd3zdIQPcIFR0MOwcbZMgsLk130_provenance.
• NP400752.RAVjF8V_JpzHPOqTq0QN9g5WZCIryUQ3oXDF-8oJUAlvg130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP400752.RAVjF8V_JpzHPOqTq0QN9g5WZCIryUQ3oXDF-8oJUAlvg130_provenance.
• NP512169.RAJEcVPIkAOW4n0HMu3YYyHoX1f57pSW6IEPHelwNy-Kc130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP512169.RAJEcVPIkAOW4n0HMu3YYyHoX1f57pSW6IEPHelwNy-Kc130_provenance.
• NP615304.RAOKTqmjNG_4949kQYpDDITBzsvCpquDhhll5xrvJ35BU130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP615304.RAOKTqmjNG_4949kQYpDDITBzsvCpquDhhll5xrvJ35BU130_provenance.
• assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP357189.RAsUpSrRZy8Yh5TYYx7AxWOs-N6anVn2zkONzldV5j4aE130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP357189.RAsUpSrRZy8Yh5TYYx7AxWOs-N6anVn2zkONzldV5j4aE130_provenance.
• NP662691.RArsRIdyJ0RnXwg3HchWYBwVjNpbovpTgySdcEBZYwD1w130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP662691.RArsRIdyJ0RnXwg3HchWYBwVjNpbovpTgySdcEBZYwD1w130_provenance.
• NP537858.RAhsg0Mowf78ubVOXd0TtCL1QTcakmWNtXQLQFe0vaa3k130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP537858.RAhsg0Mowf78ubVOXd0TtCL1QTcakmWNtXQLQFe0vaa3k130_provenance.
• NP357055.RAzUAuFhDLQrizpi2uNrsarDWGHRc0_5R9iORF64dWuRk130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP357055.RAzUAuFhDLQrizpi2uNrsarDWGHRc0_5R9iORF64dWuRk130_provenance.
• NP662723.RAGfBYMpK_3s8blvSFPNM9ZuHXc7FaLYHDS0_q63wLH0A130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP662723.RAGfBYMpK_3s8blvSFPNM9ZuHXc7FaLYHDS0_q63wLH0A130_provenance.
• NP537861.RAOGgbavhNoayFrqN6lyYO2xJLWXmPpnZBvkMhVX6A3AA130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP537861.RAOGgbavhNoayFrqN6lyYO2xJLWXmPpnZBvkMhVX6A3AA130_provenance.
• NP537859.RA1FFRmk31Noq1B1OGtn2wNhwTJ5fZrzIwqzxDEkmkIsM130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP537859.RA1FFRmk31Noq1B1OGtn2wNhwTJ5fZrzIwqzxDEkmkIsM130_provenance.
• NP537860.RA9_yNyPceVUXMfp0AAGJdGxWAh3XVGRg7u60_S41cn5g130_assertion description "[We demonstrate that 1) FGFR1 endocytosis is defective in the VHL mutant and is rescued by reexpression of wild-type VHL, 2) VHL is recruited to FGFR1-containing, but not EGFR-containing, endosomal vesicles, 3) VHL exhibits a functional relationship with Rab5a and dynamin 2 in FGFR1 internalization, and 4) the endocytic function of VHL is mediated through the metastasis suppressor Nm23, a protein known to regulate dynamin-dependent endocytosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP537860.RA9_yNyPceVUXMfp0AAGJdGxWAh3XVGRg7u60_S41cn5g130_provenance.