Matches in Nanopublications for { ?s ?p "[We further explored the link between disrupted FGF endothelial signaling and progression of atherosclerosis by introducing endothelial-specific deletion of FGF receptor substrate 2 ? (Frs2a) in atherosclerotic (Apoe(-/-)) mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
Showing items 1 to 4 of
4
with 100 items per page.
- assertion description "[We further explored the link between disrupted FGF endothelial signaling and progression of atherosclerosis by introducing endothelial-specific deletion of FGF receptor substrate 2 ? (Frs2a) in atherosclerotic (Apoe(-/-)) mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[We further explored the link between disrupted FGF endothelial signaling and progression of atherosclerosis by introducing endothelial-specific deletion of FGF receptor substrate 2 ? (Frs2a) in atherosclerotic (Apoe(-/-)) mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP1295342.RAUxtxg3fLKqcEcH97ybeMq3xXRiocfNT3RsJsiBTcBgA130_assertion description "[We further explored the link between disrupted FGF endothelial signaling and progression of atherosclerosis by introducing endothelial-specific deletion of FGF receptor substrate 2 ? (Frs2a) in atherosclerotic (Apoe(-/-)) mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1295342.RAUxtxg3fLKqcEcH97ybeMq3xXRiocfNT3RsJsiBTcBgA130_provenance.
- NP1295340.RAgLyxuR9zncUJqIfTHHBiuXTeV5iBe7Bt3GFP4UDEipo130_assertion description "[We further explored the link between disrupted FGF endothelial signaling and progression of atherosclerosis by introducing endothelial-specific deletion of FGF receptor substrate 2 ? (Frs2a) in atherosclerotic (Apoe(-/-)) mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1295340.RAgLyxuR9zncUJqIfTHHBiuXTeV5iBe7Bt3GFP4UDEipo130_provenance.