Matches in Nanopublications for { ?s ?p "[We further found that miR-26a exerted its antiangiogenesis function, at least in part, by inhibiting HGF-hepatocyte growth factor receptor (cMet) and its downstream signaling pathway, in turn, suppressing VEGFA production in HCC cells and impairing VEGFR2-signaling in endothelial cells.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP1130078.RAeBvn5NhW3-0GHlRa0GrvqqU4is341sTN2wlci19h2ng130_assertion description "[We further found that miR-26a exerted its antiangiogenesis function, at least in part, by inhibiting HGF-hepatocyte growth factor receptor (cMet) and its downstream signaling pathway, in turn, suppressing VEGFA production in HCC cells and impairing VEGFR2-signaling in endothelial cells.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1130078.RAeBvn5NhW3-0GHlRa0GrvqqU4is341sTN2wlci19h2ng130_provenance.
- NP554547.RAjQujC3nyZyRPsLNfWD8DgTOCJFAmJiMQ596fV9nBGtc130_assertion description "[We further found that miR-26a exerted its antiangiogenesis function, at least in part, by inhibiting HGF-hepatocyte growth factor receptor (cMet) and its downstream signaling pathway, in turn, suppressing VEGFA production in HCC cells and impairing VEGFR2-signaling in endothelial cells.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP554547.RAjQujC3nyZyRPsLNfWD8DgTOCJFAmJiMQ596fV9nBGtc130_provenance.