Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP854531.RABJsqjqpBmFPy63nN_C4sXzsCLHMrYc2J2GxvLWtsQVg130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP854531.RABJsqjqpBmFPy63nN_C4sXzsCLHMrYc2J2GxvLWtsQVg130_provenance.
• NP651249.RAsgG3sfSnZAWnsJyfoask1wyxfeKQzclVD1z7oR0skYM130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP651249.RAsgG3sfSnZAWnsJyfoask1wyxfeKQzclVD1z7oR0skYM130_provenance.
• NP478966.RAaLj1zkTAr52ma2421tjwY1hFzdbXWJiq82yA7jIFw8I130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP478966.RAaLj1zkTAr52ma2421tjwY1hFzdbXWJiq82yA7jIFw8I130_provenance.
• NP715471.RAYnA6IWjThypFO09akXiaCVkOHSrPf43luvG8cxY9-wk130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP715471.RAYnA6IWjThypFO09akXiaCVkOHSrPf43luvG8cxY9-wk130_provenance.
• assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
• NP874605.RAdh8yU-RMafvVSli2tpQ1p3A9ZPJ3InSMdWWb3DAoWy4130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP874605.RAdh8yU-RMafvVSli2tpQ1p3A9ZPJ3InSMdWWb3DAoWy4130_provenance.
• NP479017.RA7nRkMaujs7_r9fyi6MEZ8Vpq2fKDyDcKbYVgkutM1SA130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP479017.RA7nRkMaujs7_r9fyi6MEZ8Vpq2fKDyDcKbYVgkutM1SA130_provenance.
• NP874604.RAJYpR0QF39ExFcs27D8uuG4-T-_emW1ymO10uQzjrRCg130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP874604.RAJYpR0QF39ExFcs27D8uuG4-T-_emW1ymO10uQzjrRCg130_provenance.
• NP715487.RA92NHH8c7zYs1LNlYjyPC7U19T2RHmqVKO22PS1u9DYw130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP715487.RA92NHH8c7zYs1LNlYjyPC7U19T2RHmqVKO22PS1u9DYw130_provenance.
• NP874603.RA868_qOu6ics7ojWRMSGeoer470K3bY4Djfzf5nEBSWY130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP874603.RA868_qOu6ics7ojWRMSGeoer470K3bY4Djfzf5nEBSWY130_provenance.
• NP874606.RA4uq12ik_FYr9754Mq62X1UKIF-wc3M4AJK64WR2VBUg130_assertion description "[With the established roles of IFN? and TLRs in atherosclerotic pathology, the STAT1-dependent signal integration between IFN? and TLR in ECs and VSMCs in response to exogenous and endogenous atherogenic ligands could result in amplification of pro-inflammatory responses in the damaged vessel and be a novel mechanism involved in the initiation and progression of atherosclerosis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP874606.RA4uq12ik_FYr9754Mq62X1UKIF-wc3M4AJK64WR2VBUg130_provenance.