Matches in Nanopublications for { ?s ?p " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." ?g. }
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- _2 value " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." provenance.
- _4 value " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." provenance.
- _2 value " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." provenance.
- _4 value " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." provenance.
- _4 value " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." provenance.
- _4 value " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." provenance.
- _2 value " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." provenance.
- _2 value " 3.3. The p38 MAPK in senescence and tumor suppression The description of a possible p38 MAPK/p21/HBP1 pathway coincides with several reports for possible roles of p38 MAPK in senescence and tumor suppression. Senescence is characterized as an irreversible cell cycle arrest that occurs upon extended passage of cells in culture. The process of senescence can be triggered by telomere shortening, H2O2 exposure, or chronic Ras stimulation in primary cells." provenance.