Matches in Nanopublications for { ?s ?p "Additional evidence of the involvement of these inflammatory pathways is supported by the protective effect of some anti-inflammatory drugs against obesity-induced insulin resistance. Aspirin may inhibit not only IKK and JNK (93,94) but other Ser/Thr kinases (mamalian target of rapamycin and protein kinase B/Akt) related to TNF-alpha insulin resistance by phosphorylation of IRS-1 at serine residues (94). Furthermore, through its antioxidant properties, aspirin has been shown to reduce the activation of NF-kappaB or AP-1 associated with reactive oxygen species (95)." ?g. }
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- _3 value "Additional evidence of the involvement of these inflammatory pathways is supported by the protective effect of some anti-inflammatory drugs against obesity-induced insulin resistance. Aspirin may inhibit not only IKK and JNK (93,94) but other Ser/Thr kinases (mamalian target of rapamycin and protein kinase B/Akt) related to TNF-alpha insulin resistance by phosphorylation of IRS-1 at serine residues (94). Furthermore, through its antioxidant properties, aspirin has been shown to reduce the activation of NF-kappaB or AP-1 associated with reactive oxygen species (95)." provenance.
- _3 value "Additional evidence of the involvement of these inflammatory pathways is supported by the protective effect of some anti-inflammatory drugs against obesity-induced insulin resistance. Aspirin may inhibit not only IKK and JNK (93,94) but other Ser/Thr kinases (mamalian target of rapamycin and protein kinase B/Akt) related to TNF-alpha insulin resistance by phosphorylation of IRS-1 at serine residues (94). Furthermore, through its antioxidant properties, aspirin has been shown to reduce the activation of NF-kappaB or AP-1 associated with reactive oxygen species (95)." provenance.