Matches in Nanopublications for { ?s ?p "Furthermore, real-time quantitative PCR indicated that a subset of Wnt family genes was highly expressed in the gut mesenchyme of Foxl1-deficient mice compared with that of wild-type mice. A previous study has demonstrated that Foxl1 is involved in the regulation of the beta-catenin signaling pathway (26). We also confirmed that the nuclear beta-catenin translocation was enhanced in the small intestinal epithelium of Foxl1?/? mice by Western blot analysis (Fig. 5). " ?g. }
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- _4 value "Furthermore, real-time quantitative PCR indicated that a subset of Wnt family genes was highly expressed in the gut mesenchyme of Foxl1-deficient mice compared with that of wild-type mice. A previous study has demonstrated that Foxl1 is involved in the regulation of the beta-catenin signaling pathway (26). We also confirmed that the nuclear beta-catenin translocation was enhanced in the small intestinal epithelium of Foxl1?/? mice by Western blot analysis (Fig. 5). " provenance.
- _4 value "Furthermore, real-time quantitative PCR indicated that a subset of Wnt family genes was highly expressed in the gut mesenchyme of Foxl1-deficient mice compared with that of wild-type mice. A previous study has demonstrated that Foxl1 is involved in the regulation of the beta-catenin signaling pathway (26). We also confirmed that the nuclear beta-catenin translocation was enhanced in the small intestinal epithelium of Foxl1?/? mice by Western blot analysis (Fig. 5). " provenance.
- _4 value "Furthermore, real-time quantitative PCR indicated that a subset of Wnt family genes was highly expressed in the gut mesenchyme of Foxl1-deficient mice compared with that of wild-type mice. A previous study has demonstrated that Foxl1 is involved in the regulation of the beta-catenin signaling pathway (26). We also confirmed that the nuclear beta-catenin translocation was enhanced in the small intestinal epithelium of Foxl1?/? mice by Western blot analysis (Fig. 5). " provenance.
- _4 value "Furthermore, real-time quantitative PCR indicated that a subset of Wnt family genes was highly expressed in the gut mesenchyme of Foxl1-deficient mice compared with that of wild-type mice. A previous study has demonstrated that Foxl1 is involved in the regulation of the beta-catenin signaling pathway (26). We also confirmed that the nuclear beta-catenin translocation was enhanced in the small intestinal epithelium of Foxl1?/? mice by Western blot analysis (Fig. 5). " provenance.