Matches in Nanopublications for { ?s ?p "Furthermore, with the use of a tetracycline-regulated expression system, it has been demonstrated that VEGF withdrawal can force newly formed tumor blood vessels into regression (6). Finally, in vitro studies have demonstrated that VEGF mediates this survival function for endothelial cells through VEGFR-2 and its downstream signal transduction pathways (7), suggesting that interference with VEGF/VEGFR-2 signaling may exert not only anti-angiogenic but also anti-vascular efficacy." ?g. }
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- _3 value "Furthermore, with the use of a tetracycline-regulated expression system, it has been demonstrated that VEGF withdrawal can force newly formed tumor blood vessels into regression (6). Finally, in vitro studies have demonstrated that VEGF mediates this survival function for endothelial cells through VEGFR-2 and its downstream signal transduction pathways (7), suggesting that interference with VEGF/VEGFR-2 signaling may exert not only anti-angiogenic but also anti-vascular efficacy." provenance.
- _3 value "Furthermore, with the use of a tetracycline-regulated expression system, it has been demonstrated that VEGF withdrawal can force newly formed tumor blood vessels into regression (6). Finally, in vitro studies have demonstrated that VEGF mediates this survival function for endothelial cells through VEGFR-2 and its downstream signal transduction pathways (7), suggesting that interference with VEGF/VEGFR-2 signaling may exert not only anti-angiogenic but also anti-vascular efficacy." provenance.