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- _6 value "However, when we analyzed the long-term inhibitory effect of insulin on dex/cAMP-induced PEPCK and G6Pase mRNAs, substantial differences were found. Whereas insulin was able to suppress gluconeogenic gene expression for 12 h in wild-type neonatal hepatocytes, this effect was prolonged up to 36 h in PTP1B-deficient cells (Fig. 9BGo)." provenance.
- _3 value "However, when we analyzed the long-term inhibitory effect of insulin on dex/cAMP-induced PEPCK and G6Pase mRNAs, substantial differences were found. Whereas insulin was able to suppress gluconeogenic gene expression for 12 h in wild-type neonatal hepatocytes, this effect was prolonged up to 36 h in PTP1B-deficient cells (Fig. 9BGo)." provenance.
- _6 value "However, when we analyzed the long-term inhibitory effect of insulin on dex/cAMP-induced PEPCK and G6Pase mRNAs, substantial differences were found. Whereas insulin was able to suppress gluconeogenic gene expression for 12 h in wild-type neonatal hepatocytes, this effect was prolonged up to 36 h in PTP1B-deficient cells (Fig. 9BGo)." provenance.
- _3 value "However, when we analyzed the long-term inhibitory effect of insulin on dex/cAMP-induced PEPCK and G6Pase mRNAs, substantial differences were found. Whereas insulin was able to suppress gluconeogenic gene expression for 12 h in wild-type neonatal hepatocytes, this effect was prolonged up to 36 h in PTP1B-deficient cells (Fig. 9BGo)." provenance.