Matches in Nanopublications for { ?s ?p "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " ?g. }
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- _5 value "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " provenance.
- _3 value "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " provenance.
- _5 value "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " provenance.
- _5 value "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " provenance.
- _3 value "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " provenance.
- _5 value "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " provenance.
- _5 value "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " provenance.
- _5 value "ITSN-1 knockdown acts as a proapoptotic signal that causes mitochondrial outer membrane permeabilization, dissipation of the mitochondrial membrane potential, and generation of reactive oxygen species.These effects were secondary to decreased activation of Erk1/2 and its direct activator MEK. " provenance.