Matches in Nanopublications for { ?s ?p "In addition, we (12) and others (13,14) have previously demonstrated that TNF-A represses the mRNA levels of an adipocyte-derived hormone, ACRP30, which inhibits gluconeogenesis in the liver (15), and decreases plasma levels of glucose (16) and FFAs (17). Thus, the increased circulating FFA concentrations and/or reduced ACRP30 protein levels could mediate some of the delayed effects of TNF-A on whole-body energy metabolism." ?g. }
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- _3 value "In addition, we (12) and others (13,14) have previously demonstrated that TNF-A represses the mRNA levels of an adipocyte-derived hormone, ACRP30, which inhibits gluconeogenesis in the liver (15), and decreases plasma levels of glucose (16) and FFAs (17). Thus, the increased circulating FFA concentrations and/or reduced ACRP30 protein levels could mediate some of the delayed effects of TNF-A on whole-body energy metabolism." provenance.
- _3 value "In addition, we (12) and others (13,14) have previously demonstrated that TNF-A represses the mRNA levels of an adipocyte-derived hormone, ACRP30, which inhibits gluconeogenesis in the liver (15), and decreases plasma levels of glucose (16) and FFAs (17). Thus, the increased circulating FFA concentrations and/or reduced ACRP30 protein levels could mediate some of the delayed effects of TNF-A on whole-body energy metabolism." provenance.
- _3 value "In addition, we (12) and others (13,14) have previously demonstrated that TNF-A represses the mRNA levels of an adipocyte-derived hormone, ACRP30, which inhibits gluconeogenesis in the liver (15), and decreases plasma levels of glucose (16) and FFAs (17). Thus, the increased circulating FFA concentrations and/or reduced ACRP30 protein levels could mediate some of the delayed effects of TNF-A on whole-body energy metabolism." provenance.
- _3 value "In addition, we (12) and others (13,14) have previously demonstrated that TNF-A represses the mRNA levels of an adipocyte-derived hormone, ACRP30, which inhibits gluconeogenesis in the liver (15), and decreases plasma levels of glucose (16) and FFAs (17). Thus, the increased circulating FFA concentrations and/or reduced ACRP30 protein levels could mediate some of the delayed effects of TNF-A on whole-body energy metabolism." provenance.